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Titlebook: Endometriosis; Pathogenesis and Tre Tasuku Harada Book 2014 Springer Japan 2014 Endometriosis.infertility.oocytes.ovarian cysts.ovaries.pro

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發(fā)表于 2025-3-21 19:02:06 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書(shū)目名稱Endometriosis
副標(biāo)題Pathogenesis and Tre
編輯Tasuku Harada
視頻videohttp://file.papertrans.cn/310/309824/309824.mp4
概述Helps clarify pathogenesis and pathophysiology of endometriosis.Covers all essential information for clinicians, including prevention and practical treatment.Written by nearly 40 internationally renow
圖書(shū)封面Titlebook: Endometriosis; Pathogenesis and Tre Tasuku Harada Book 2014 Springer Japan 2014 Endometriosis.infertility.oocytes.ovarian cysts.ovaries.pro
描述.Endometriosis is one of the most common gynecological diseases, causing?pelvic pain, infertility, and cancer. The condition reduces the quality of life for the estimated 10% of menstruating women who are affected by it.?This book provides a clear insight into?the pathogenesis and pathophysiology of endometriosis—essential?information for?clinicians?intending to successfully treat the condition. The first part covers a range of topics including stem cells, NK cells, macrophages, inflammation, cytokines, epigenetics, prostaglandin, sex steroids, aromatase, apoptosis, nerve fibers, and malignant transformation to aid in understanding the pathogenesis and pathophysiology of the disease. The second part provides useful tools for therapy, specifically, notable recent techniques in prevention, imaging, biomarkers, medical treatments, surgical treatments, prevention of recurrence, and infertility treatments. Each chapter was written by an expert in the field, with nearly 40 researchers and clinicians contributing to the work. This book will be highly useful to clinicians as well as to research scientists who are interested in this enigmatic disease.?? .
出版日期Book 2014
關(guān)鍵詞Endometriosis; infertility; oocytes; ovarian cysts; ovaries; prostaglandin
版次1
doihttps://doi.org/10.1007/978-4-431-54421-0
isbn_softcover978-4-431-56157-6
isbn_ebook978-4-431-54421-0
copyrightSpringer Japan 2014
The information of publication is updating

書(shū)目名稱Endometriosis影響因子(影響力)




書(shū)目名稱Endometriosis影響因子(影響力)學(xué)科排名




書(shū)目名稱Endometriosis網(wǎng)絡(luò)公開(kāi)度




書(shū)目名稱Endometriosis網(wǎng)絡(luò)公開(kāi)度學(xué)科排名




書(shū)目名稱Endometriosis被引頻次




書(shū)目名稱Endometriosis被引頻次學(xué)科排名




書(shū)目名稱Endometriosis年度引用




書(shū)目名稱Endometriosis年度引用學(xué)科排名




書(shū)目名稱Endometriosis讀者反饋




書(shū)目名稱Endometriosis讀者反饋學(xué)科排名




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發(fā)表于 2025-3-21 21:26:20 | 只看該作者
Lektüren – literarischer Horizonts triggered primarily by bacterial endotoxin (lipopolysaccharide, LPS) and is mediated by Toll-like receptor 4 (TLR4) and showed their involvement in the growth regulation of visible peritoneal endometriosis. Even with the careful eye of an expert surgeon, we may sometimes miss to detect peritoneal
板凳
發(fā)表于 2025-3-22 02:01:59 | 只看該作者
https://doi.org/10.1007/978-1-349-12501-2n eutopic endometrium during the menstrual cycle. HLA-G expression was only identified in eutopic endometrium during the menstrual phase, but not during the proliferative or secretory phases. HLA-G-expressing cells were also detected in peritoneal fluid during the menstrual phase..Retrograde menstru
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發(fā)表于 2025-3-22 04:49:45 | 只看該作者
Authority, Power and the State, 1916–20s. Thus, unveiling the molecular mechanisms that underlie macrophage dysfunctions is a critical area of research, which would lead to the development of novel medical treatments for endometriosis. In this chapter, we described how macrophages can play a critical role in the pathophysiology of endome
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Where to Obtain Tools and Parts,r molecules may act as a neurotrophic factor in endometriosis. Increased numbers of nerve fibres, increased amount of neurotrophins and different types of nerve fibres in endometriosis may explain why women with endometriosis experience pain.
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發(fā)表于 2025-3-22 22:03:11 | 只看該作者
Where to Obtain Tools and Parts,s most closely resemble the disease in women; however, rodent models are more cost effective and readily available. Nevertheless, since rodents do not menstruate, rodent models used for the research in endometriosis have certain limitations. We addressed the question whether a murine endometriosis m
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