Titlebook: Endocrinology of the Vasculature; James R. Sowers Book 1996 Humana Press Inc. 1996 Diabetes.Diabetes mellitus.growth factor.insulin.signal

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Effects of Low Density Lipoproteins on Endothelial Cell Signaling and Adhesion Molecule Expressionlular free calcium concentration [Ca.]. is an important mediator of this activation . Hypercholesterolemia, particularly the low-density lipoprotein (LDL) fraction, is an important risk factor for the development of atherosclerosis, a disease process intimately involving endothelial cells . LDL stim

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Angiotensin II Signal Transduction in Vascular Smooth Musclewater balance, blood pressure, and vascular tone. All is produced both systemically and locally in the vessel wall by the actions of renin, which converts angiotensinogen into AI, and angiotensinconverting enzyme (ACE), which cleaves AI to form AII. In addition to its vasoconstrictor activity, AII h

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The Vascular Renin System a cascade of angiotensin peptides including angiotensin (A) I, II, and III. AII, the most prominent of these peptide hormones, initiates through specific plasma membrane receptors, a host of physiological actions ranging from vasoconstriction to volume and growth regulation .. Circulating plasma le

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Effects of Insulin on Vascular Smooth Muscle Contraction in patients with essential hypertension, obesity, and non-insulin-dependent diabetes mellitus . It has been proposed that compensatory hyperinsulinemia increases sympathetic nerve activity and renal Na reabsorption, which could contribute to the genesis ofhypertension . Despite the effects of hyper

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Sex Hormones and the Vasculatureof cardiovascular disease is lower in premenopausal women than in age-matched men, but rises steadily after menopause (2). Postmenopausal women on estrogen replacement therapy have less severe coronary artery disease and a lower risk for cardiovascular mortality than women without hormone treatment

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https://doi.org/10.1007/978-3-642-66435-9 fashion on adjacent vascular smooth muscle cells (VSMC) to activate soluble guanylyl cyclase and generate cyclic guanosine 3’,5’ monophosphate (cGMP) . cGMP, in turn, mediates many of NO-mediated actions in VSMC. However, a number of cGMP-independent effects have been observed following NO stimulat

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