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Titlebook: Endocrinology of Pregnancy; Fuller W. Bazer Book 1998 Springer Science+Business Media New York 1998 Uterus.birth.endocrinology.fetus.growt

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樓主: Ingrown-Toenail
31#
發(fā)表于 2025-3-27 00:42:02 | 只看該作者
Endocrine Control of Trophoblast-Uterine Epithelial Cell Interactions,l stroma following trophoblast penetration of uterine epithelium (invasive hemochorial and endotheliochorial implantation), but in others, stromal invasion does not occur, and the intimate association between uterine epithelium and conceptus tissues is retained (noninvasive epitheliochorial implanta
32#
發(fā)表于 2025-3-27 03:45:17 | 只看該作者
33#
發(fā)表于 2025-3-27 06:36:20 | 只看該作者
Secretion and Metabolism of Steroids in Primate Mammals During Pregnancy, Estrogen also may stimulate utero-placental blood flow to provide nutrients and oxygen to the placenta and fetus for growth and development. Estrogen has been implicated in the cascade of events that initiate the process of labor. Finally, cortisol produced by the fetal adrenal gland has a critical
34#
發(fā)表于 2025-3-27 12:12:54 | 只看該作者
Placental Lactogen and Growth Hormone,velopment ., whereas in hypophysectomized rats lobulo-alveolar growth is stimulated with exogenous PRL .. The difficulty in defining the requisite roles for GH and PRL during pregnancy at least in part results from the fact that during pregnancy placental-derived members of the GH/PRL gene family ar
35#
發(fā)表于 2025-3-27 15:30:21 | 只看該作者
36#
發(fā)表于 2025-3-27 19:45:13 | 只看該作者
37#
發(fā)表于 2025-3-28 01:25:02 | 只看該作者
2523-3785 pregnancy. This book will become the standard reference source not only for reproductive scientists, but also for those clinicians who want better to understand the complex factors that affect pregnancy-and their pregnant patients. ..978-1-4612-7291-5978-1-4612-1804-3Series ISSN 2523-3785 Series E-ISSN 2523-3793
38#
發(fā)表于 2025-3-28 02:37:33 | 只看該作者
39#
發(fā)表于 2025-3-28 06:23:22 | 只看該作者
40#
發(fā)表于 2025-3-28 12:24:49 | 只看該作者
https://doi.org/10.1007/978-3-319-67900-6ear transcription factor receptor. Induced null mutations in the genes for the estrogen (ERKO) and progesterone receptor (PRKO) in mice, support the view that both estradiol-17β (E.) and P. act directly in the uterus. In both null mutants, a uterus develops; however, it is estrogen-insensitive and h
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