Titlebook: Endocrine Disorders in Thalassemia; Physiopathological a Sebastiano Andò (Head of Endocrine Physiopathology Book 1995 Springer-Verlag Berli

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A Multicenter Study on Endocrine Disorders in Thalassemia — Introductionarea and spans through the Middle East, to India and East Asia. In 1988 the WHO reported that 240,000 children per year are born with a major hemoglobinopathy — 20% with thalassemias and 80% with the sickle cell syndrome. The total number of thalassemic patients transfused and followed up at Italian

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Endocrine Pathology in Thalassemiareased absorption from the gastrointestinal tract, with deleterious effects on internal organs and tissues [2, 7–9]. With modern treatment and longer survival, endocrine dysfunction assumes greater importance [1, 3–5]. This study was carried out in an attempt to correlate such dysfunction with patho

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Endocrine Complications in Cyprian Thalassemic Patientsblood transfusions results in deposits in many organs including the endocrine glands, causing endocrine dysfunction. The incidence of endocrinopathies in thalassemics has been reported to be high by several investigators [2, 3]. The gland most sensitive to iron deposition is thought to be the pituit

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Growth in Homozygous β-Thalassemic Patientsriety of endocrine abnormalities frequently occur in such patients. A number of studies have shown that as thalassemic patients approach the age of puberty, a percentage develop growth retardation [1]. The growth failure of these patients has been attributed to hypothyroidism, delayed sexual maturat

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Relationship Between GH Response to Stimuli, Levels of IGF-1, and Final Height are still major problems. Growth failure is generally accepted as beginning at the age of 10, with a marked decline in the growth rate after the age of 12–14 years [1, 2]. A recent report shows that height impairment occurs before the age of 10 [3]. While it is possible that delayed puberty may be

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Glucose Intolerance and Diabetes in Thalassaemia majorn a particular tissue exceeds the tissue or blood binding capacity of iron, and large quantities of free nontransferrin iron appear. This “free iron” is a catalyst of the production of oxygen species that damage cells and peroxidize membrane lipids, leading to cell destruction. The liver has a large

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A Multicenter Study on Endocrine Disorders in Thalassemia — Introductionroach to endocrine complications in thalassemia as too sophisticated. For this reason, I am certain that all of us are grateful to the organizing comittee for having planned this International Mediterranean Conference and for giving us an opportunity to collect and share the experiences from differe

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Endocrine Complications in Cyprian Thalassemic Patientspendent diabetes mellitus (IDDM) after the age of 15 years. The frequency of hypoparathyroidism and hypothyroidism is also very low, so that in this report we will address the issues of growth failure and hypogonadism, which are more commonly observed.

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https://doi.org/10.1007/978-1-349-66019-3n a particular tissue exceeds the tissue or blood binding capacity of iron, and large quantities of free nontransferrin iron appear. This “free iron” is a catalyst of the production of oxygen species that damage cells and peroxidize membrane lipids, leading to cell destruction. The liver has a large

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