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Titlebook: Endocrine Aspects of Successful Aging: Genes, Hormones and Lifestyles; Philippe Chanson,Jacques Epelbaum,Yves Christen Book 2004 Springer-

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樓主: 揭發(fā)
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發(fā)表于 2025-3-28 18:31:43 | 只看該作者
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發(fā)表于 2025-3-28 22:03:53 | 只看該作者
Estrogen and Cognitive Functioning in Women,trogen on brain structure and function (discussed in detail in other chapters of this volume). It is now clear that this sex hormone exerts effects on brain morphology and on brain chemistry that provide biological plausibility for the hypothesis that estrogen could enhance and/or preserve aspects o
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發(fā)表于 2025-3-28 23:31:32 | 只看該作者
Genetic Polymorphisms and Age-Related Diseases The Example of Osteoporosis,s identified a number of risk factors for these common age-related disorders. Examples include obesity for diabetes, low bone mineral density for osteoporosis, and smoking for cancer and cardiovascular disease. Some of these risk factors are used in clinical practice, for example to identify at-risk
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發(fā)表于 2025-3-29 03:10:20 | 只看該作者
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發(fā)表于 2025-3-29 09:31:25 | 只看該作者
Testosterone Supplementation and Aging-associated Sarcopenia,therapies increase muscle mass and that increased muscle mass would be associated with improvements in measures of muscle performance and physical function. We assume that improved physical function in humans with sarcopenia will translate into decreased risk of disability, falls, and fractures and
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發(fā)表于 2025-3-29 14:06:26 | 只看該作者
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發(fā)表于 2025-3-29 23:07:21 | 只看該作者
Prediction of Death in Elderly Men: Endocrine Factors,se and disease-related disability, high cognitive and physical functional capacity, and active engagement with life. This definition was taken as a starting point in a study among 403 independently living elderly men. Since low muscle strength and functional ability were highly predictive of four-ye
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發(fā)表于 2025-3-30 03:05:19 | 只看該作者
Research and Perspectives in Endocrine Interactionshttp://image.papertrans.cn/e/image/309701.jpg
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發(fā)表于 2025-3-30 05:41:49 | 只看該作者
Asaf Romirowsky,Alexander H. Joffee of neurodegenerative diseases. Increased transcription mediates the increase of GFAP expression. We hypothesize that increased GFAP expression is a factor in age-related impairments of synaptic plasticity, and we are developing an in vitro model to test this hypothesis.
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