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Titlebook: Eicosanoids and Radiation; Peter Polgar Book 1988 Kluwer Academic Publishers, Boston 1988 cancer.cancer research.cell.cell culture.hormone

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發(fā)表于 2025-3-21 17:14:25 | 只看該作者 |倒序瀏覽 |閱讀模式
書目名稱Eicosanoids and Radiation
編輯Peter Polgar
視頻videohttp://file.papertrans.cn/304/303156/303156.mp4
叢書名稱Prostaglandins, Leukotrienes, and Cancer
圖書封面Titlebook: Eicosanoids and Radiation;  Peter Polgar Book 1988 Kluwer Academic Publishers, Boston 1988 cancer.cancer research.cell.cell culture.hormone
出版日期Book 1988
關(guān)鍵詞cancer; cancer research; cell; cell culture; hormones; metastasis
版次1
doihttps://doi.org/10.1007/978-1-4613-1723-4
isbn_softcover978-1-4612-8970-8
isbn_ebook978-1-4613-1723-4
copyrightKluwer Academic Publishers, Boston 1988
The information of publication is updating

書目名稱Eicosanoids and Radiation影響因子(影響力)




書目名稱Eicosanoids and Radiation影響因子(影響力)學(xué)科排名




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發(fā)表于 2025-3-21 22:57:13 | 只看該作者
Radiobiology of Blood Vessels,the irradiated field. The timing and severity of the acute effects depend upon : (1) transit time for maturing cells within the tissue compartment; (2) dose fraction size; and (3) protraction (the total time in which the radiation is delivered).
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地板
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https://doi.org/10.1057/9781403943743the irradiated field. The timing and severity of the acute effects depend upon : (1) transit time for maturing cells within the tissue compartment; (2) dose fraction size; and (3) protraction (the total time in which the radiation is delivered).
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發(fā)表于 2025-3-22 14:43:03 | 只看該作者
The Developing Countries Today,ry overproduction of reticulin fibers, hyperplasia of elastic fibers eventuating in elastosis, increased levels of the glycosaminoglycans (GAGs) comprising the ground substance and changes in the microvasculature. First described in actinically damaged humans (4,5), systematic investigation required
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發(fā)表于 2025-3-22 22:32:58 | 只看該作者
https://doi.org/10.1057/9781137463661platelet aggregatory (TXA2, PGG2 and PGH2) (10–14) and anti-aggregatory properties (PGE2 and PGI2) (15–18) may play a role in radiation-induced tissue injury. This hypothesis is based on the observation that radiation exposure results in tissue injury (19–35) and tissue injury is associated with the release of cyclooxygenase products (36–39).
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發(fā)表于 2025-3-23 04:13:43 | 只看該作者
Hilary Footitt,Angela M. Crack,Wine Tesseurf knowledge which has accumulated about eicosanoids and their important role as mediators of cutaneous inflammation, including UV-induced cutaneous changes. In this chapter, we will review the known effects of UV radiation on cutaneous eicosanoid metabolism,
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