Titlebook: Ecstasy: The Clinical, Pharmacological and Neurotoxicological Effects of the Drug MDMA; Stephen J. Peroutka Book 1990 Kluwer Academic Publ

Truman

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AMEND

coddle

Testing Psychotherapies and Drug Therapies: The Case of Psychedelic Drugs,tle attention has been given to the possibility of using drugs directly to enhance the process of psychotherapy — fortifying the therapeutic alliance and facilitating the production of memories, fantasies, and insights. A change may now be coming; for example, a psychiatrist known for his research o

alcohol-abuse

Recreational Use of MDMA, that MDMA may be one of the most widely used recreational drugs of the late 1980s. Alternatively, MDMA use may be restricted to a few locations and may be relatively inconsequential in comparison to drugs such as cocaine and marijuana. There are simply no good data on which to adequately define and

發(fā)生

Human Deaths and Toxic Reactions Attributed to MDMA and MDEA,ble media attention as recreational drugs popular among college students and young professionals. MDMA, more commonly known as “Ecstasy,” has been available on the illicit drug market since 1968 (1). with increasing popularity in the late 1970s and early 1980s. On the other hand, MDEA, also known as

Oligarchy

The Public Health Implications of MDMA Use,en the born-again protagonists who say that once you have tried it you will see the light and will defend it against any attack, and there have been the staunch antagonists who say this is nothing but LSD revisited and it will certainly destroy our youth (1).

Oligarchy

Structure-Activity Relationships of MDMA and Related Compounds: A New Class of Psychoactive Agents?lass named entactogens [1–3]. In this chapter evidence will be presented to support this, through a discussion of the data acquired in efforts directed toward testing this hypothesis. Although these studies are far from complete, the results gathered thus far, together with those from other laborato

憤世嫉俗者

Neurochemical Effects of MDMA,ethamphetamine, given in large repeated doses (10–15 mg/kg, s.c., every six hours for five doses), caused a dose-related decrease in tyrosine hydroxylase (TH) activity in the neostriatum [1, 2] and substantia nigra [3]. A parallel decline in concentrations of dopamine (DA) and its metabolites, dihyd

neutral-posture

植物茂盛