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Titlebook: Early Life Origins of Health and Disease; E. Marelyn Wintour,Julie A. Owens Book 2006 The Editor(s) (if applicable) and The Author(s), und

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書目名稱Early Life Origins of Health and Disease
編輯E. Marelyn Wintour,Julie A. Owens
視頻videohttp://file.papertrans.cn/301/300754/300754.mp4
概述Presents a very up-to date and readable summary of our current understanding of the importance of life before birth, from conception to delivery, and in infancy and childhood, on the subsequent health
叢書名稱Advances in Experimental Medicine and Biology
圖書封面Titlebook: Early Life Origins of Health and Disease;  E. Marelyn Wintour,Julie A. Owens Book 2006 The Editor(s) (if applicable) and The Author(s), und
描述.Early Life Origins of Health and Disease is a new book which presents and discusses the many factors that may have impact on normal development. In a concise and readable manner, the authors consider both the proven and suggestive evidence that the high prevalence of hypertension, diabetes, obesity and, in some populations, kidney disease, may not be all due to genetics or adult environment alone. There is good evidence that stress and more subtle dietary deficiencies, as well as placental malfunction, may increase the risk that the offspring will develop these problems in later life. Finally, new and emerging evidence for other areas of human health and disease such a motor control and mental health is critically reviewed for the first time. The book is a ‘must’ for all scientists interested in researching these areas, as there is a critical evaluation of the methodology used and suggestions for the ‘optimal’ way in which to investigate these phenomena. .
出版日期Book 2006
關(guān)鍵詞Diabetes; Imprinting; amino acids; development; embryo; genetics; growth; health; implantation; nutrition; obe
版次1
doihttps://doi.org/10.1007/0-387-32632-4
isbn_softcover978-1-4419-3950-0
isbn_ebook978-0-387-32632-0Series ISSN 0065-2598 Series E-ISSN 2214-8019
issn_series 0065-2598
copyrightThe Editor(s) (if applicable) and The Author(s), under exclusive license to Springer Science+Busines
The information of publication is updating

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Developmental Programming of Cardiovascular Dysfunctionsimilarity with the human data and may imply an important role in the aetiology of developmentally induced cardiovascular risk. Studies of inflammatory bio-markers, lipid profiles and compliance in animal models are too few to allow comparison. Increasing evidence for altered sympathetic activity in
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Programming of Obesity—Experimental Evidenceetal programming recent experimental studies suggest that leptin and insulin resistance are critical endocrine defects in the pathogenesis of programming-induced obesity and metabolic disorders. Recent work has also shown that offspring of undernourished mothers display reduced locomotor activity in
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