Titlebook: E. coli; Shiga Toxin Methods Dana Philpott,Frank Ebel Book 2003 Humana Press 2003

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Analysis of Pathogenicity Islands of STEC,tected by polymeraase chain reaction (PCR) or hybridization, which also allows the identification of attenuated pathogenic strains. These might have lost not only one virulence gene but a whole set thereof that is part of a mobile genetic element.

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Generation of Isogenic Deletion Mutants of STEC,inactivate a particular gene, we use a precise and convenient method that is based on the generation of an in-frame deletion within this gene. The advantages of this technique in comparison with others (e.g., transposon mutagenesis or site-directed insertional mutagenesis) are as follows:

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Methods for the Identification of Host Receptors for Shiga Toxin,differentiation antigen (.). Tissue localization of Gb. is associated with sites of Stx-induced pathology in animal models (.–.) and in humans (.) and, as such, is a major determinant of Stx1-induced disease.

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Methods for Detection of STEC in Humans, perspective. In the outbreak setting, rapid diagnosis of cases and immediate notification of health authorities is essential for effective epidemiological intervention. Early diagnosis also creates a window of opportunity for therapeutic intervention. Agents capable of adsorbing and neutralizing fr

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STEC in the Food Chain,to hemorrhagic colitis (HC), hemolytic uremic syndrome (HUS), and thrombotic thrombocytopenic purpura (TTP). In addition to the protopathotype O157:H7, other enterohemorrhagic . (EHEC) serovars (e.g., O26:H11, O103:H2, O111:H., O145:H., and O157:H., have caused severe infections in humans (.). Nearl

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