Titlebook: Dopamine in the CNS II; Gaetano Chiara Book 2002 Springer-Verlag Berlin Heidelberg 2002 Behavioural Psychology.Cell Biology.Central Nervou

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尾巴

Dopamine and Depression, think and act more slowly, and experience particular difficulty in initiating actions (. 1985; . and . 1992). A simple hypothesis to explain both of these central features of depression is that they reflect an impairment of incentive motivation. Incentives are stimuli associated with rewards, which

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Atypical Antipsychotics,hown between their affinity for D. receptors and dosages used for treating positive symptoms of schizophrenia (. et al. 1976; . et al. 1976). However, a direct relationship also exists between blockade of D. receptors and the induction of extrapyramidal symptoms and the elevation of plasma prolactin

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https://doi.org/10.1007/978-3-658-19662-2decreased by activation of D2 receptors (. et al. 1979). This opposite role of the two types of DA receptors is not ubiquitous, as they have also been shown to act in a cooperative manner in several models (see . 1989 for review). For example, the two subtypes act synergistically to promote locomoto

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https://doi.org/10.1007/978-3-658-19662-2DA: its prominent role in drug-reward. In fact, drug-induced stimulation of DA transmission is itself rewarding (. and . 1987; . 1995). This circumstance, while acting as a powerful incentive of studies on the role of DA in drug-reinforcement and drug-addiction, has been also utilized as the basis f

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FinTech-L?sungen für den MittelstandWhile additional classifications of symptoms have been proposed (e.g., . 1980; . 1982; . 1987; . et al. 1988, 1999; . et al. 1989; . 1990; . and . 1994; . et al. 1995; . 1995), the dopamine (DA) hypothesis of schizophrenia has been primarily related to the positive-negative classification (see below

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Den Kontext der Organisation bestimmen,hown between their affinity for D. receptors and dosages used for treating positive symptoms of schizophrenia (. et al. 1976; . et al. 1976). However, a direct relationship also exists between blockade of D. receptors and the induction of extrapyramidal symptoms and the elevation of plasma prolactin