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Titlebook: Diseases of the Thyroid; Lewis E. Braverman Book 19971st edition Humana Press 1997 birth.hormone.hormones.pathophysiology.physiology.pregn

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發(fā)表于 2025-3-21 18:38:48 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書目名稱Diseases of the Thyroid
編輯Lewis E. Braverman
視頻videohttp://file.papertrans.cn/282/281328/281328.mp4
叢書名稱Contemporary Endocrinology
圖書封面Titlebook: Diseases of the Thyroid;  Lewis E. Braverman Book 19971st edition Humana Press 1997 birth.hormone.hormones.pathophysiology.physiology.pregn
描述Nationally and internationally recognized clinicians and researchers combine the latest understanding of thyroid physiology and pathophysiology with the most up-to-date clinical knowledge to help clinical endocrinologists and primary care physicians understand and treat a wide variety of thyroid disorders from birth to old age. Topics range from neonatal thyroid screening, thyroid dysfunction during infancy and childhood, peripartum thyroid disorders, and thyroid disease in the elderly to the pathogenesis and treatment of nodular goiter, thyroid cancer, thyrotoxicosis, and hypothyroidism. The book takes advantage of the latest clinical thinking and of the newest findings of the molecular biology of thyroid hormone action.
出版日期Book 19971st edition
關(guān)鍵詞birth; hormone; hormones; pathophysiology; physiology; pregnancy; primary care; thyroid hormone
版次1
doihttps://doi.org/10.1007/978-1-4757-2594-0
isbn_ebook978-1-4757-2594-0Series ISSN 2523-3785 Series E-ISSN 2523-3793
issn_series 2523-3785
copyrightHumana Press 1997
The information of publication is updating

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Molecular Mechanisms of Nuclear Thyroid Hormone Action,on. TH then enters the nucleus where it binds to the nuclear thyroid hormone receptor (TR) with high affinity and specificity (K.s in the nanomolar range). TR is a ligand-regulated transcription factor that is intimately associated with chromatin, and also associates with additional nuclear proteins
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Resistance to Thyroid Hormone,rated that genetic mutations of hormone receptor proteins or proteins involved in the signal transduction are the underlying cause of most clinical conditions that we prefer today to call not “pseudo,” but “resistance to thyroid hormone action.”
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Dynamics of Environmental Repletion of Iodine, difficulties, cost, suspicions by some people that the supplements contained contraceptives, and the preference of the population for their traditional source, rock salt from the desert floor, which is low in iodine. Faced with these difficulties, we devised a method of providing iodine to the popu
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Iodine-Induced Thyroid Disease,nction even in the presence of iodine excess. When large amounts of iodine are given to subjects with normal thyroid function, a transient decrease in the synthesis of the thyroid hormones occurs for approx 48 h. This acute inhibitory effects of iodine on thyroid hormone synthesis is termed the acut
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