Titlebook: Diseases of the Oral Mucosa; Study Guide and Revi Enno Schmidt Textbook 2021 The Editor(s) (if applicable) and The Author(s), under exclusi

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Integrated Series in Information SystemsPC-K6a, PC-K6b, PC-K6c, PC-K16 and PC-K17 according to the mutated gene are transmitted in an autosomal dominant way with occurrence of spontaneous mutations in about 30% of cases. Main clinical features include nail thickening (pachyonychia) and subungual hyperkeratosis, focal palmoplantar keratode

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Business Meets Community in Virtual Berlin oral aphthae with spontaneous improvement within 3–10?days. It usually begins in the second decade and decreases in severity with age. The aphtha is a shallow oval or round mucosal ulcer covered by a greyish-white or yellow pseudomembrane and surrounded by a red halo. There are three types of RAS,

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https://doi.org/10.1007/978-3-319-22069-7aphthous ulcers, genital ulcers, erythema nodosum, and papulopustular lesions are the most common findings. Besides, signs and symptoms related to many organ systems such as ocular, skeleton, vascular, gastrointestinal, and central nervous system can be seen. It is prevalent in the Far East and arou

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https://doi.org/10.1007/978-3-319-22069-7t primarily affects older, mostly white women. Clinically, CUS patients exhibit erosive or ulcerative lesions of oral mucosa resembling erosive lichen planus and/or desquamative gingivitis. Routine histology may exhibit features of lichenoid mucositis and is non-diagnostic. Direct immunofluorescence

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Gayathri R. Devi Ph.D.,Sritama Nathlthough oral lichen sclerosus is an exceedingly rare condition. Oral lesions are porcelain-white or whitish, well-demarcated maculae or plaques. The most common sites are labial mucosa and lips, buccal mucosa, and gingiva. Most of the lesions are asymptomatic. However, a third of patients have sympt

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https://doi.org/10.1007/978-1-4939-3625-0y for which numerous therapeutic options were administered with various success rates. A plethora of differential diagnoses have to be taken into account, altering also possible therapeutic interventions. Therefore, orofacial granulomatosis remains an exclusion diagnosis. The scope of this chapter i

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Gayathri R. Devi Ph.D.,Sritama Nathtoantibodies interfering with the desmosomal cell-cell adhesion molecules desmoglein 1 and 3. The oral cavity is the main affected area and the initial site to be involved in most patients with PV. Clinical symptoms include enanthema, blisters, and partly painful erosions and ulcers localized anywhe

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https://doi.org/10.1007/978-3-642-22354-9 which is further divided into pemphigus diseases with autoantibodies to keratinocyte cell surfaces and pemphigoid diseases with autoantibodies to epidermal basement membrane zone. Paraneoplastic pemphigus (PNP) shows polymorphous mucocutaneous lesions, and is associated with either benign or malign