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Titlebook: Diseases of the Arterial Wall; Jean-Pierre Camilleri (Professeur),Colin L. Berry Book 1989 Springer-Verlag Berlin Heidelberg 1989 aneurys

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發(fā)表于 2025-3-21 19:45:21 | 只看該作者 |倒序瀏覽 |閱讀模式
書目名稱Diseases of the Arterial Wall
編輯Jean-Pierre Camilleri (Professeur),Colin L. Berry
視頻videohttp://file.papertrans.cn/282/281293/281293.mp4
圖書封面Titlebook: Diseases of the Arterial Wall;  Jean-Pierre Camilleri (Professeur),Colin L. Berry  Book 1989 Springer-Verlag Berlin Heidelberg 1989 aneurys
出版日期Book 1989
關(guān)鍵詞aneurysm; arteries; atherosclerosis; biology; blood vessel; cardiovascular; endothelium; hypertension; molec
版次1
doihttps://doi.org/10.1007/978-1-4471-1464-2
isbn_softcover978-1-4471-1466-6
isbn_ebook978-1-4471-1464-2
copyrightSpringer-Verlag Berlin Heidelberg 1989
The information of publication is updating

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沙發(fā)
發(fā)表于 2025-3-21 22:14:49 | 只看該作者
Extracellular Matrix of the Arterial Vessel Wallnd the position in the arterial tree of the vessel. Nevertheless, the major components of the vessel wall extracellular matrices are collagens of various types and elastin. The cohesion between cells and these fibrous proteins is ensured by glycoproteins such as fibronectin and laminin and by proteo
板凳
發(fā)表于 2025-3-22 01:19:14 | 只看該作者
Organogenesis of the Arterial Wallremove waste from developing tissues. The subsequent differentiation and development of the organism eventually produces a specialized cardiovascular system with distinct variations in form in different parts. In between embryonic and adult life vascular systems will have developed, involuted, been
地板
發(fā)表于 2025-3-22 08:28:14 | 只看該作者
Effects of “Ageing” on Arterial Cells in Vitroof a thickening of the intima caused by the proliferation of smooth-muscle cells which have migrated from the media towards the intima [60,61]. According to certain investigators, this stimulation of the growth of smooth-muscle cells, which are usually quiescent in normal arteries, could be explaine
5#
發(fā)表于 2025-3-22 12:11:56 | 只看該作者
Structural Modifications of the Arterial Wall in Hypertensionficiency, suggest that the level of blood pressure is related directly to both primary functions of the cardiovascular system: the peripheral blood flow and filtration pressure in capillaries necessary for oxygen delivery to tissues [8,15]. For example, in spontaneously hypertensive rats, these two
6#
發(fā)表于 2025-3-22 16:53:27 | 只看該作者
7#
發(fā)表于 2025-3-22 19:38:00 | 只看該作者
The Arterial Wall and the Haemostatic Processtivated by the vessel itself. Any alteration in the endothelium transforms the vascular wall into a thrombogenic surface to which blood platelets will adhere, and triggers the multistep process of haemostatis in which both platelets and plasma are involved. This phenomenon is beneficial because it l
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發(fā)表于 2025-3-23 01:15:14 | 只看該作者
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發(fā)表于 2025-3-23 03:13:35 | 只看該作者
Atherosclerosis: Cellular Aspects mysterious. In the last few years, however, significant progress has been made in understanding the roles played by arterial wall components, blood-borne cells and plasma constituents in the development of the atheromatous plaque [for reviews see 4,43,49,63,90,92,93,130]. This process appears more
10#
發(fā)表于 2025-3-23 08:36:06 | 只看該作者
Extracellular Matrix Changes in Atherosclerosis accumulation of lipid and extracellular matrix, calcification and thrombosis. Although the initiating factors are still not completely understood, it is clear that the deposited extracellular matrix affects all of these events. There are two fundamental questions concerning the role of the extracel
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