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Titlebook: Dietary Fats, Lipids, Hormones, and Tumorigenesis; New Horizons in Basi David Heber,David Kritchevsky Book 1996 The Editor(s) (if applicabl

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發(fā)表于 2025-3-21 19:13:45 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書(shū)目名稱Dietary Fats, Lipids, Hormones, and Tumorigenesis
副標(biāo)題New Horizons in Basi
編輯David Heber,David Kritchevsky
視頻videohttp://file.papertrans.cn/279/278576/278576.mp4
叢書(shū)名稱Advances in Experimental Medicine and Biology
圖書(shū)封面Titlebook: Dietary Fats, Lipids, Hormones, and Tumorigenesis; New Horizons in Basi David Heber,David Kritchevsky Book 1996 The Editor(s) (if applicabl
描述This book was inspired by a gatheringofscientists in Los Angeles in 1994 under the auspices of the UCLA Clinical Nutrition Research Unit which is funded by the National Cancer Institute to promote new research into nutrition and cancer prevention. This unit supports research integrating basic and metabolic/clinical investigations which examine observations from epidemiologic studies and their application to the prevention ofcommon forms ofcancer through nutritional intervention. There is a great deal ofinformation from epidemiologic, experimental and metabolic studies implicating elements ofthe diet as important in the development and progression of common forms ofcancer including breast cancer, colon cancer, prostate cancer, and uterine cancer. When these forms ofcancerareexaminedcarefully, it isclearthat they share anumber ofcommon etiologic factors related to dietary fat, lipids, and hormones. A human cancer is usually discovered at a point where it has formed a detectable mass. For many forms of cancer, this may require 10 to 15 years from the time when the cancer is first initiated. Nutritional efforts at prevention may delay the progression ofcancer to a detectable mass resul
出版日期Book 1996
關(guān)鍵詞Colon; Lipid; Nutrition; Oxidation; Vitamin D; cancer; metabolism
版次1
doihttps://doi.org/10.1007/978-1-4613-1151-5
isbn_softcover978-1-4612-8450-5
isbn_ebook978-1-4613-1151-5Series ISSN 0065-2598 Series E-ISSN 2214-8019
issn_series 0065-2598
copyrightThe Editor(s) (if applicable) and The Author(s), under exclusive license to Springer Science+Busines
The information of publication is updating

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Examining Supply Chains from Practiceed in many common forms of cancer has also been shown to translate environmental influences into excess adiposity (5). Many hormones involved in obesity also play a role in the initiation and promotion of cancer both at a cellular, paracrine, and systemic level (6). For example, reproductive hormone
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https://doi.org/10.1007/978-3-642-56604-2leic acid and its product arachidonic acid, and a negative association with use of a drug blocking its metabolism to eicosanoids. Another potentially important factor in arachidonate metabo?lism is variation in its endogenous distribution. We have recently reported abnormal distribution of arachidon
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Quality in Chemical Measurementspment of certain cancers as suggested by the association between dietary fat intake and breast cancer risk in many epidemiological studies. The mechanism by which dietary and hormonal factors increase the risk of development of certain cancers is not known. Whether modification of dietary fat intake
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Einstieg und erste Schritte in QMF,gh alterations in methyl metabolism has been somewhat neglected. Recent reports indicating an increased risk of cancer and precancerous lesions of various types in humans associated with low folate intakes or low red blood cell folate levels increase the relevance of such investigations (Giovannucci
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發(fā)表于 2025-3-22 20:07:26 | 只看該作者
Quantitative Models for Reverse Logisticslogy of pancreatic cancer. Families at high risk for pancreatic cancer have been reported. Cigarette smoking is a risk factor, but the strength of the association is much less than in lung cancer. Animal studies have implicated several chemical exposures in pancreatic carcinogenesis. High dietary fa
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發(fā)表于 2025-3-23 01:57:07 | 只看該作者
Overview of Fat and Calories in Tumorigenesis,m heated fat was considerably more tumorigenic (70% incidence at 6 months) then oxidized fat (38%) or irradeated fat (40%). The intact fat (hydrogenated vegetable oil) led to 31% incidence of skin tumors. Their work led Miller et al. (4) to compare the effects of different fats on the carcinogenicit
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