Titlebook: Diet — Brain Connections; Impact on Memory, Mo Mark P. Mattson Book 2002 Springer Science+Business Media New York 2002 Alzheimer.aging.beha

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Dietary Neurotoxins, by toxic substances naturally occurring in some foods, or as a result of contaminants or residues of various kind. Some of these agents have neurotoxic properties and may contribute to the etiology of certain psychiatric disorders or neurodegenerative diseases. This chapter focuses on a selected nu

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Dietary Anti-Oxidants and the Risk for Brain Disease: The Hypothesis and Epidemiologic Evidence,or dementia in late-age. Anti-oxidant supplements have been proposed as secondary or primary interventions against dementia. A brief overview is given of the hypotheses relating anti-oxidants to the major forms of dementia, Alzheimer’s disease and vascular dementia. The existing observational and ra

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,Copper, Zinc, and Alzheimer’s Disease,nce is then presented for altered Cu or Zn metabolism or physiology in Alzheimer’s disease, the major age-dependent cause of memory loss in man. Altered Cu and Zn metabolism or physiology in AD is likely to be directly coupled to pathogenesis, but may not be directly linked to diet.

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e benefits of widely used dietary supplements including creatine and Gingko biloba; .-contribution of dietary toxins such as metals and pesticides to neurological disorders..978-1-4613-5378-2978-1-4615-1067-3

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The Principles of Quantum Theoryconditions in which LTP is attenuated will be discussed and the hypothesis that the proinflammatory cytokine interleukin-1β (IL-1β) induces oxidative stress will be explored..Synaptic plasticity describes the ability of interacting neurons to modulate their Connectivity in response to a particular S

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https://doi.org/10.1007/978-3-540-49972-5ome are improved by caloric restriction in animal models of Alzheimer’s, Parkinson’s and Hungtington’s diseases, and stroke. Caloric restriction may promote synaptic plasticity and thereby improve cognitive and motor performance, and can stimulate neurogenesis (production of new neurons from stem ce

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