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Titlebook: Diet and Cancer; Markers, Prevention, Maryce M. Jacobs Book 1994 The Editor(s) (if applicable) and The Author(s), under exclusive license t

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發(fā)表于 2025-3-21 20:06:23 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書(shū)目名稱Diet and Cancer
副標(biāo)題Markers, Prevention,
編輯Maryce M. Jacobs
視頻videohttp://file.papertrans.cn/279/278559/278559.mp4
叢書(shū)名稱Advances in Experimental Medicine and Biology
圖書(shū)封面Titlebook: Diet and Cancer; Markers, Prevention, Maryce M. Jacobs Book 1994 The Editor(s) (if applicable) and The Author(s), under exclusive license t
描述The American Institute for Cancer Research (AICR) sponsored its third annual confer- ence on nutrition and cancer. The theme was "Diet and Cancer: Markers, Prevention, and Treatment. " The conference was held October 29-30, 1992 at the Ritz Carlton Hotel in McLean, Virginia. This proceedings contains chapters from the platform presentations and abstracts from the poster presentations. Several chapters address each of four session topics: Retinoids as Differentiation Agents in Cancer Therapy, Biological Markers of Cancer Risk, Chemoprevention of Cancer by Non-Nutrients in Foods, and Nutritional Problems and Support in the Treatment of Cancer. The first three chapters discuss in detail different mechanisms by which retinoids influence differentiation and provide evidence to support their use in cancer therapy. In vivo and in vitro studies show the effects of retinoic acid (RA) on tumorigenicity and cellu- lar/molecular events. A synopsis of data showing the involvement of the retinoblastoma (RB) gene in HL-60 cell differentiation induced by RA and 1,2S-dihydroxy vitamin D3 (VD) is presented. In SCID (severe combined immunodeficiency) mice injected with HL-60 3 human leukemia cells an
出版日期Book 1994
關(guān)鍵詞Carcinom; Colon; DNA; Translation; Vitamin A; Vitamin D; cancer; gene expression; proteins
版次1
doihttps://doi.org/10.1007/978-1-4899-0939-8
isbn_softcover978-1-4899-0941-1
isbn_ebook978-1-4899-0939-8Series ISSN 0065-2598 Series E-ISSN 2214-8019
issn_series 0065-2598
copyrightThe Editor(s) (if applicable) and The Author(s), under exclusive license to Springer Science+Busines
The information of publication is updating

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Retinoic Acid Suppresses Human Papillomavirus Type 16 (HPV16)-Mediated Transformation of Human Kerathe carotenoids, and cancer risk at several epithelial sites,. including the cervix.. Retinoids also may be effective in treating premalignant lesions such as cervical intraepithelial neoplasia (CIN).. In fact, phase I and phase II clinical trials of all-.-retinoic acid (RA) for CIN have shown that R
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Cancer Chemoprevention by Green Tea Componentsthat block carcinogenesis in animal tumor bioassay systems.. For these reasons, changes in dietary habits with the routine intake of more cancer chemopreventive agents, specifically those which have shown preventive effects in a wide range of animal tumor bioassay systems, are an attractive approach
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Carcinogen-DNA and Protein Adducts as Intermediate Biomarkers for Human Chemoprotection Trialssure levels to toxic agents, markers of effect indicate a biological response to an exposure, and markers of susceptibility provide information about the inherent sensitivity of an individual to a toxic agent. By definition some of these markers are chemical-agent specific, such as a carcinogen-DNA
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Altered Expression of Intermediate Biomarkers for Mammary Preneoplasia: Relevance to Cancer Chemopreti-estrogens, and indole derivatives resulted in downregulation of all the three perturbed biomarkers. Enhanced expression of molecular, endocrine, or cellular markers in initiated, tumorigenic target cells, and their . modulation by prototypic tumor suppressing agents demonstrates that the biomarke
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