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Titlebook: Dictionary of Rheumatology; Jozef Rovensky,Juraj Payer Book 20091st edition Springer-Verlag Vienna 2009 Arthritis.Dictionary.Osteology.Rhe

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樓主: 監(jiān)督
11#
發(fā)表于 2025-3-23 09:45:01 | 只看該作者
Space, Models and Geometric FantasiesMovement of extremities of the body towards the midline.
12#
發(fā)表于 2025-3-23 15:05:21 | 只看該作者
α1-microglobulin (α1M)A protein synthesised in the liver and present in blood, serum and urine. Complexes of α.M with monomeric immunoglobulin A (IgA) participate in renal IgA nephropathy where the serum level of α.M is also usually increased.
13#
發(fā)表于 2025-3-23 18:36:54 | 只看該作者
AbataceptAbatacept (Orencia.) is an injectable, synthetic (man-made) soluble fusion protein that consists of the extracellular domain of human cytotoxic T-lymphocyte-associated antigen 4 (CTLA-4) linked to the modified Fc portion of human immunoglobulin G1 (IgG1). Abatacept is produced by recombinant DNA technology in a mammalian cell expression system.
14#
發(fā)表于 2025-3-23 22:35:35 | 只看該作者
AbductionMovement of extremities of the body away from the body’s midline.
15#
發(fā)表于 2025-3-24 04:14:03 | 只看該作者
16#
發(fā)表于 2025-3-24 08:24:53 | 只看該作者
Action potentialElectrical activity in the nerve axon or muscle fibre according to the “all or nothing” type, when the membrane potential polarity ceases or is restored abruptly.
17#
發(fā)表于 2025-3-24 13:00:10 | 只看該作者
Acute febrile neutrophilic dermatosis (Sweet’s syndrome)A rare disease of unknown aetiology characterised by marked inflammatory neutrophilic skin infiltrates.
18#
發(fā)表于 2025-3-24 18:43:30 | 只看該作者
AdductionMovement of extremities of the body towards the midline.
19#
發(fā)表于 2025-3-24 21:07:28 | 只看該作者
Achilles tendon bursa. Tendonitis can be part of the clinical picture of spondyloarthritis, especially ankylosing spondylitis. It can remain thickened (by fibrosis) after the inflammation has subsided and nodules may be palpable.
20#
發(fā)表于 2025-3-25 03:05:53 | 只看該作者
Achondroplasia and hypochondroplasiat growth factor receptor-3 (FGFR-3). It belongs to the most frequent dysplasias with an incidence of 1 to 15.000–77.000 births, depending on race. The mutation of FGFR-3 results in a defect of chondrocyte proliferation in the growth cartilage of long bones.
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