Titlebook: Diagnosis and Therapy of Porphyrias and Lead Intoxication; International Sympos Manfred Doss Conference proceedings 1978 Springer-Verlag Be

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刀鋒

Differential Patterns of Heme and Prophyrin Biosynthesis in Liver and Kidney: Metal Ion Blockade of r the most part, is due to the ready induction response of liver δ-aminolevulinate synthetase (ALAS) — the rate-limiting enzyme of porphyrin-heme synthesis — to the exogenous chemicals and the physiologic factors that alter porphyrin and heme production. Compounds known to be highly potent in alteri

寒冷

斜坡

Permissive Effects of Hormones on the Induction of δ-Aminolevulinic Acid Synthase in Cultured Chick-s enzyme is present in the mitochondria and is inducible in the liver by a variety of compounds of hydrophobic nature. Studies of the regulation of ALA-S have been facilitated by the introduction of a tissue-culture technique for cultivating monolayer of chick-embryo liver cells (2). A major drawbac

parasite

刪減

A New Approach to Porphyriaine, has been very helpful for their diagnosis (8). Consecutive biochemical and clinical findings subdivided each group: erythropoietic porphyrias on the basis of different porphyrins occurring in the blood, hepatic porphyrias on the basis of porphyrins and precursors in urine, feces, and liver (8)

刪減

The Clinical Chemistry of Variegate Porphyria with Special Reference to the Identification of a New (8), with an estimated incidence of approximately 9000 affected persons. How this remarkable situation has come about has been fully documented by Dean (5) in his monograph, which makes it clear that virtually all South African variegate prophyrics are descendants of a daughter of a marriage that t

Glucose

Expression of the Gene Defects of Acute Intermittent Porphyria (AIP) and Erythropoietic Protoporphyric cells (7) reflects the primary gene defect of AIP, and accounts for the excessive urinary excretion of porphobilinogen and 6-aminolevulinic acid (ALA). The decreased activity of ferrochelatase found in cultured skin fibroblasts and bone-marrow cells from patients with erythropoietic protoporphyri

恭維

seroma

A Case of Acute Intermittent Porphyria, Relapsing Acute Pancreatitis, and Unconjugated Hyperbilirubi The previously healthy 36-year-old woman was admitted to the hospital because of severe colicky pain in the right lower abdomen, vomiting, constipation, and red colored urine. A highly increased excretion of porphobilinogen in the urine and a normal fecal excretion of porphyrins was compatible with