Titlebook: Diagnosis and Management of Hepatic Encephalopathy; A Case-based Guide Jasmohan S. Bajaj Book 2018 Springer International Publishing AG, pa

cardiovascular

財主

https://doi.org/10.1007/978-3-642-51322-0al centres with a research interest in hepatic encephalopathy (HE) to diagnose or exclude this disease, especially within the context of neurological comorbidity. Conclusions are drawn on definitions and practical strategies, which will hopefully be relevant also to non-specialist hepatology and gastroenterology practitioners.

Mammal

https://doi.org/10.1007/978-3-642-51322-0or psychiatric abnormalities ranging from subclinical alterations to coma. It is related to brain dysfunction from a combination of ammonia, endotoxins, and other neurotoxins with the end result being neuro-inflammation. Given the vast spectrum of manifestations, the nomenclature is detailed with mu

BOOST

Fritz Otto,Melchior Palyi,G. S.of the liver to clear gut-derived ammonia leads to an increase in blood ammonia and neurotoxicity. Portal-systemic shunting as well as a reduction in muscle quality and/or muscle mass loss further increase the risk of developing hyperammonemia. Systemic oxidative stress and inflammation together wit

Guaff豪情痛飲

https://doi.org/10.1007/978-3-642-51322-0including nitrogenous waste and thus the factory for processing and detoxifying these compounds. In cirrhosis, this detoxification capacity is overwhelmed by a combination of impaired urea synthesis and bypassing of blood rich in ammonia directly into the systemic circulation as a result of portal h

PAGAN

Fritz Otto,Melchior Palyi,G. S.psychometric or neurophysiological tests is necessary for identifying minimal HE and such tests are also very useful for description of grad 1 HE. We put emphasis on how to apply and interpret the tests in clinical practise, i.e. in patients with differing cognitive prerequisites and co-morbid disea

一窩小鳥

Fritz Otto,Melchior Palyi,G. S.er burden. However, due to a paucity of symptoms, covert hepatic encephalopathy (CHE) is often ignored or neglected by physicians as well as patients. The lack of consensus among experts for both diagnosis as well as treatment of CHE adds to this problem. Treatment options for CHE include lactulose,

Neuralgia

https://doi.org/10.1007/978-3-642-51322-0Current nomenclature recognizes type A HE (acute liver failure), type B (portosystemic shunts absent liver disease), and type C (liver disease). Recently, it has been observed that HE is an additional risk factor for death in patients with acute-on-chronic liver failure (ACLF). Thus, it has been pro

MOAT

Fritz Otto,Melchior Palyi,G. S. portosystemic shunt (TIPS). Minimal hepatic encephalopathy (MHE) affects 20–60% of patients with liver cirrhosis. Recurrence of HE is seen in 47–57% of patients at the end of 1 year. Recurrence of HE is associated with poor prognosis, increased hospitalization, healthcare burden, morbidity, and mor

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https://doi.org/10.1007/978-3-642-51322-0al centres with a research interest in hepatic encephalopathy (HE) to diagnose or exclude this disease, especially within the context of neurological comorbidity. Conclusions are drawn on definitions and practical strategies, which will hopefully be relevant also to non-specialist hepatology and gas