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Titlebook: Diabetic Nephropathy; Pathophysiology and Joris J. Roelofs,Liffert Vogt Book 2019 Springer International Publishing AG, part of Springer N

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41#
發(fā)表于 2025-3-28 15:19:19 | 只看該作者
are addressed...Diabetic Nephropathy.?will be of interest for nephrologists, diabetologists, internists, transplant physicians, public health professionals, basic scientists, geneticists, epidemiologists, pathologists, and molecular and cell biologists working in the field of diabetes and its complications..978-3-030-06678-9978-3-319-93521-8
42#
發(fā)表于 2025-3-28 19:58:49 | 只看該作者
43#
發(fā)表于 2025-3-28 23:18:25 | 只看該作者
https://doi.org/10.1007/978-3-531-18970-3uminuric DN phenotype; African-Americans and South Asians are also more likely to progress rapidly to more advanced stages of CKD. Factors such as genetic predisposition, differing access to standard DM care, as well as suboptimal DM-related health-care spending are identified drivers for observed d
44#
發(fā)表于 2025-3-29 05:00:37 | 只看該作者
45#
發(fā)表于 2025-3-29 08:43:43 | 只看該作者
Handbuch P?dagogische AnthropologieThen, we move to the natural history of DKD progression and relationships between structural changes, renal dysfunction, and diabetes duration. Finally, reversal of DKD lesions following long-term normoglycemia after pancreas transplantation and animal models of DKD are discussed.
46#
發(fā)表于 2025-3-29 14:58:37 | 只看該作者
47#
發(fā)表于 2025-3-29 16:14:19 | 只看該作者
48#
發(fā)表于 2025-3-29 22:32:52 | 只看該作者
49#
發(fā)表于 2025-3-30 00:08:27 | 只看該作者
Jürgen Straub,Pradeep Chakkarathgulation is believed to disrupt the metabolism of NAD., diminishing the replenishment of the glomerulus with nicotinamide mononucleotide?(NMN) from the proximal tubule; this results in Sirt1 downregulation and tight junction protein claudin-1 upregulation in glomerular podocytes. Intracellular nicot
50#
發(fā)表于 2025-3-30 06:07:17 | 只看該作者
Methoden der Film- und Fernsehanalysecollectively play a role in the development and progression of interstitial fibrosis. A comprehensive understanding of the underlying mechanisms and downstream pathways involved in the tubulointerstitial fibrosis is mandatory to develop groundbreaking therapeutic options and novel clinical strategie
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