Titlebook: Deprenyl — Past and Future; W. Kuhn,P. Kraus,H. Przuntek Conference proceedings 1996 Springer-Verlag/Wien 1996 Alzheimer.Parkinson.diagnos

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Conference proceedings 1996rkinsonian drug called budipine with various modes of action also influencing altered non dopaminergic systems in Parkinson‘s disease. It is evident, that many questions on the cause, course and treatment of Parkinson‘s disease are still unanswered and therefore the ideal way to treat a parkinsonian patient remains to be defined.

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Manfred Steiner,Sebastian Schneiderntiapoptotic molecules which promote cell survival. Further delineation of the precise mechanism whereby selegiline induces this effect may permit for the development of enhanced neuroprotective benefits in PD patients.

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Die Business School im E-Zeitalterd l-methamphetamine have some qualitatively different actions than their disomer counterparts on EEG and cognitive functioning which might result in beneficial clinical effects and complement beneficial clinical actions of l-deprenyl itself.

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Bewertung von E-Business Strategienon of the disease could be observed. For depression a higher dosage of deprenyl between 30 to 60 mg/day appears to be necessary for effective treatment. No positive results were found in amyotrophic lateral sclerosis and in tardive dyskinesias.

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The clinical potential of Deprenyl in neurologic and psychiatric disorders,on of the disease could be observed. For depression a higher dosage of deprenyl between 30 to 60 mg/day appears to be necessary for effective treatment. No positive results were found in amyotrophic lateral sclerosis and in tardive dyskinesias.