Titlebook: Defects of Secretion in Cystic Fibrosis; Carsten Schultz Conference proceedings 2005 The Editor(s) (if applicable) and The Author(s), unde

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archaeology

An Inositol Phosphate Analog, INO-4995, Normalizes Electrophysiology in CF Airway Epithelia,s. These studies indicate that transient exposure to an inositol polyphosphate analog, INO-4995, causes long lasting but ultimately reversible changes in therapeutically relevant electrophysiological properties of CF human nasal epithelia. This avoids a major problem encountered with other ion chann

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Conference proceedings 2005stic fibrosis provides in depth original work as well as review material on many of the relevant physiological and molecular topics in the field. Subjects covered include the interplay of the various epithelial ion channels, the underlying intracellular signal transduction, mucus secretion, and nove

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Hatsue Ishibashi-Ueda,Keiko Ohta-Ogoat molecules the generated anhydride can attach or if the anhydride only has been hydrolyzed. Once this and other questions have been answered, one can start to address potential therapeutic approaches using recent advances in mucin knowledge.

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Biosynthesis and Secretion of Mucins, Especially the MUC2 Mucin, in Relation to Cystic Fibrosis,at molecules the generated anhydride can attach or if the anhydride only has been hydrolyzed. Once this and other questions have been answered, one can start to address potential therapeutic approaches using recent advances in mucin knowledge.

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ar biology to drug design.Presents the most recent updates i.Defects in Secretion of Cystic Fibrosis. presents an overview on current research from leading experts in North America and Europe. This update on cystic fibrosis provides in depth original work as well as review material on many of the re

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https://doi.org/10.1007/978-1-59745-325-7 in therapeutically relevant electrophysiological properties of CF human nasal epithelia. This avoids a major problem encountered with other ion channel regulators that have been advanced as potential CF treatments, limited duration of action.

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Role of CFTR and Other Ion Channels in Cystic Fibrosis,