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Titlebook: DNA Tumor Viruses; Oncogenic Mechanisms Giuseppe Barbanti-Brodano,Mauro Bendinelli,Herman Book 1995 Springer Science+Business Media New Yo

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書目名稱DNA Tumor Viruses
副標(biāo)題Oncogenic Mechanisms
編輯Giuseppe Barbanti-Brodano,Mauro Bendinelli,Herman
視頻videohttp://file.papertrans.cn/261/260207/260207.mp4
叢書名稱Infectious Agents and Pathogenesis
圖書封面Titlebook: DNA Tumor Viruses; Oncogenic Mechanisms Giuseppe Barbanti-Brodano,Mauro Bendinelli,Herman  Book 1995 Springer Science+Business Media New Yo
描述DNA tumor viruses have long been useful experimental models of carcinogenesis and have elucidated several important mechanisms of cell transformation. Re- search in recent years has shown that human tumors have a multifactorial nature and that some DNA tumor viruses may playa key role in their etiology. The aim of this book is to assess our knowledge of DNA tumor viruses by reviewing animal models, mechanisms of transformation, association with human tumors, and possi- bilities of prevention and control by vaccination. Animal models of tumor virology have contributed significantly to our under- standing of the epidemiology and pathogenesis of virus-induced tumors. Bovine papillomaviruses induce papillomas in the intestine of cattle. The papillomas undergo a transition to carcinomas in cows feeding on bracken fern, which pro- duces a toxin with radiomimetic and immunosuppressive functions. This example of cooperation between a virus and chemical carcinogens parallels the cooperative role of human papillomaviruses (HPVs) and herpes simplex virus type 2 (HSV-2) with environmental carcinogens in the pathogenesis of cervical cancer. Likewise, hepatocarcinomas appearing in woodchucks chr
出版日期Book 1995
關(guān)鍵詞Pathogene; development; protein; tumorigenesis; virus
版次1
doihttps://doi.org/10.1007/978-1-4899-1100-1
isbn_softcover978-1-4899-1102-5
isbn_ebook978-1-4899-1100-1Series ISSN 1075-1289
issn_series 1075-1289
copyrightSpringer Science+Business Media New York 1995
The information of publication is updating

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Lecture Notes in Computer Science, we consider their general characteristics, the natural history of infection, experimental transformation and oncogenicity by BKV and JCV, as well as the evidence linking BKV, JCV, and SV40 to human neoplasia.
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Artificial Intelligence and Natural Languagetern of keratin expression, cell cycle regulation, and terminal differentiation, which ultimately gives rise to aberrant cells. With some viral types, however, these benign proliferations progress into aggressive malignancies.
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Andrey Filchenkov,Lidia Pivovarova,Jan ?i?ka induce persistent infections in the infected host. Chronic, long-lasting infections with both agents frequently result in the development of liver cirrhosis and ultimately evolve to primary liver cancer in a significant proportion of virus carriers.
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Artificial Intelligence and Natural Languagecell lines,. albeit with low efficiency. HHV-6 is ubiquitous in humans, and seroprevalence in adults reaches 90%.. Infection occurs early in life, as shown by the fact that most children become HHV-6 seropositive by 2 years of age.
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