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Titlebook: DNA Tumor Viruses; James Pipas,Blossom Damania Book 2009 Springer-Verlag New York 2009 AIDS.DNA.DNA tumor viruses.Epstein-Barr Virus.HPV s

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21#
發(fā)表于 2025-3-25 06:27:18 | 只看該作者
22#
發(fā)表于 2025-3-25 09:09:05 | 只看該作者
Martin Ebers,Paloma Kr??t Tupayleiomyosarcoma, are inconsistent and obscure pathobiologically. Vaccines to prevent infection and regimens to treat IM remain under development. However, exploiting virus-specific features of the EBV malignancies therapeutically is showing progress while at the same time illuminating the biology of cancer cells and their genesis.
23#
發(fā)表于 2025-3-25 15:19:57 | 只看該作者
EBV Diseases,leiomyosarcoma, are inconsistent and obscure pathobiologically. Vaccines to prevent infection and regimens to treat IM remain under development. However, exploiting virus-specific features of the EBV malignancies therapeutically is showing progress while at the same time illuminating the biology of cancer cells and their genesis.
24#
發(fā)表于 2025-3-25 17:31:56 | 只看該作者
25#
發(fā)表于 2025-3-25 21:23:24 | 只看該作者
26#
發(fā)表于 2025-3-26 04:08:51 | 只看該作者
27#
發(fā)表于 2025-3-26 04:37:18 | 只看該作者
The Life Cycle of Human Papillomaviruses,tion. Differentiation triggers the productive phase of the viral life cycle, including viral DNA amplification, late gene expression and virion assembly. Through the actions of viral proteins, infected cells remain active in the cell cycle, creating an environment conducive to late viral events and the production of progeny virions.
28#
發(fā)表于 2025-3-26 10:00:02 | 只看該作者
Zhe Liu,Lizong Zhang,Zhihong Rao,Guisong Liuative disease after hematopoietic stem cell transplant. EBV-specific CTLs have also shown promise as therapy for other EBV-associated malignancies, but genetic manipulation of the CTL to circumvent immune evasion strategies employed by tumors may be required before CTLs can reliably eliminate tumors in immunocompetent individuals.
29#
發(fā)表于 2025-3-26 16:22:36 | 只看該作者
30#
發(fā)表于 2025-3-26 17:05:22 | 只看該作者
Artificial Intelligence and Machine Learningtion. Differentiation triggers the productive phase of the viral life cycle, including viral DNA amplification, late gene expression and virion assembly. Through the actions of viral proteins, infected cells remain active in the cell cycle, creating an environment conducive to late viral events and the production of progeny virions.
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