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Titlebook: Cytokines in Severe Sepsis and Septic Shock; Heinz Redl,Günther Schlag Book 1999 Springer Basel AG 1999 Sepsis.clinical trial.cytokine.gen

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發(fā)表于 2025-3-21 17:14:52 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書目名稱Cytokines in Severe Sepsis and Septic Shock
編輯Heinz Redl,Günther Schlag
視頻videohttp://file.papertrans.cn/243/242649/242649.mp4
叢書名稱Progress in Inflammation Research
圖書封面Titlebook: Cytokines in Severe Sepsis and Septic Shock;  Heinz Redl,Günther Schlag Book 1999 Springer Basel AG 1999 Sepsis.clinical trial.cytokine.gen
描述t Heinz Red! and Gunther Sch!ag Ludwig Boltzmann Institute for Experimental and Clinical Traumatology, Vienna, Austria The word "sepsis" derives from the Greek meaning decay or rottenness. Tradition- ally this term has been used to describe the process of infection accompanied by the host‘s systemic inflammatory response. Based on that understanding, previous clin- ical studies have been designed to include only patients with positive blood cultures [1, 2]. However, the frequent occurrence of a septic response without the demon- stration of microorganisms in the circulation has led to a new definition and under- standing of sepsis, mainly as the systemic response of the host to an often unde- tectable microbiological or non-microbiological process [3]. The general consensus is that cytokines are central to the inflammatory response, particularly in sepsis. It is now known that not only Gram-negative but also Gram- positive, viral, and fungal infections initiate the complex cascades of cytokine release. Probably the most important aspect of bacterial action is the release of toxic bacterial products. In particular endotoxin from Gram-negative bacteria (see chap- ter by Schade) and s
出版日期Book 1999
關(guān)鍵詞Sepsis; clinical trial; cytokine; gene therapy; genetics; immunomodulation; inflammation; intensive care; me
版次1
doihttps://doi.org/10.1007/978-3-0348-8755-7
isbn_softcover978-3-0348-9759-4
isbn_ebook978-3-0348-8755-7Series ISSN 1422-7746 Series E-ISSN 2296-4525
issn_series 1422-7746
copyrightSpringer Basel AG 1999
The information of publication is updating

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Antiepileptic Drug Interactionsible for the loss of skeletal and fat tissue [1]. A prolonged catabolic state as found in patients with severe sepsis or severe burn injury may even cause death. This process was named autocannibalism of the human body [2], a reaction which cannot be stopped by nutrition and causes death due to nitr
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