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Titlebook: Cystic Fibrosis Methods and Protocols; William R. Skach Book 2002 Humana Press 2002

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發(fā)表于 2025-3-21 17:26:41 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書(shū)目名稱Cystic Fibrosis Methods and Protocols
編輯William R. Skach
視頻videohttp://file.papertrans.cn/243/242596/242596.mp4
概述Includes supplementary material:
叢書(shū)名稱Methods in Molecular Medicine
圖書(shū)封面Titlebook: Cystic Fibrosis Methods and Protocols;  William R. Skach Book 2002 Humana Press 2002
描述Since the cloning of the cystic fibrosis transmembrane conductance re- lator (CFTR) nearly a decade ago, cystic fibrosis (CF) research has witnessed a dramatic expansion into new scientific areas. Basic researchers, clinicians, and patients increasingly rely on fundamental techniques of genetics, molecular biology, electrophysiology, biochemistry, cell biology, microbiology, and immunology to understand the molecular basis of this complex disease. Research into the pathophysiology of CF has established numerous paradigms of ion channel dysfunction that extend from inflammation and infection in the airways of patients to basic mechanisms of protein processing and regulation in intracellular components. With these rapid advances has come an increasing need for research scientists to understand and utilize a growing array of basic laboratory tools. This volume of Methods in Molecular Medicine, Cystic Fibrosis Methods and Protocols satisfies that need by providing detailed protocols for the laboratory techniques used throughout CF research. From electrophysiology and cell biology, to animal models and gene therapy, the comprehensive set of methods covered here provide step-by-step inst
出版日期Book 2002
版次1
doihttps://doi.org/10.1385/1592591876
isbn_softcover978-1-61737-244-5
isbn_ebook978-1-59259-187-9Series ISSN 1543-1894 Series E-ISSN 1940-6037
issn_series 1543-1894
copyrightHumana Press 2002
The information of publication is updating

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William R. SkachIncludes supplementary material:
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Methods in Molecular Medicinehttp://image.papertrans.cn/d/image/242596.jpg
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Te Tuna-Whiri: The Knot of Eelse gene among a patient population. The type of mutations may vary from large deletions to single-base-pair (bp) substitutions, and different diseases may have different predominant types. For example, large deletions are often found in Duchenne muscular dystrophy (.) and truncation mutation is the p
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Introduction: Animism and Animacies,luding mucous plugging and chronic bacterial infection of the lung, represent the major cause of morbidity and mortality (.). It has long been suggested that mutations in CFTR, localized to the apical membrane of airway epithelia, lead to abnormalities in the fluid lining the airway surface. Accumul
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CFTR Mutation Detection by Multiplex Heteroduplex (mHET) Analysis on MDE Gele gene among a patient population. The type of mutations may vary from large deletions to single-base-pair (bp) substitutions, and different diseases may have different predominant types. For example, large deletions are often found in Duchenne muscular dystrophy (.) and truncation mutation is the p
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