Titlebook: Current and Future Immunosuppressive Therapies Following Transplantation; Mohamed H. Sayegh,Giuseppe Remuzzi Book 2001 Springer Science+Bu

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https://doi.org/10.1007/978-3-8274-2900-1 rejection [1–3]. The initial rationale for these studies was the capacity of OKT3 to stain all mature peripheral T cells and to completely abolish both T cell proliferation and the generation of cytotoxic T cell effectors in . mixed lymphocyte cultures [4]. Interestingly enough, this occurred in 19

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https://doi.org/10.1007/978-3-8274-2900-1block T cell responses at the time of antigen presentation following allograft transplantation in an effort to reorient the host immune response toward accommodation rather than rejection. The first biologic agents that were used for induction were polyclonal antilymphocyte preparations obtained fro

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https://doi.org/10.1007/978-3-8274-2900-1n. Over the past few decades, multiple investigators have defined series of families of adhesion molecules that provide for intimate cell-cell contact and facilitate interactions among another series of activation molecules. To this end, the process of adhesion is intimately associated with activati

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https://doi.org/10.1007/978-94-010-1005-4T cell; clinical trial; gene therapy; immunology; immunosuppression; kidney; organ; organ transplantation; t