Titlebook: Current Topics in Medical Mycology; Michael R. McGinnis Book 1988 Springer-Verlag New York Inc. 1988 fungi.immunology.infection.infections

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Keratin

Adhesion and Association Mechanisms of ,,, but in predisposed persons clinical infection can occur as the result of its opportunistic nature. In patients who are compromised immunologically and undergoing prolonged antimicrobic therapy, . can reach high numbers in the GI tract and, subsequently, pass through the intestinal mucosa to initia

forestry

Peptide Transport in ,: Implications for the Development of Antifungal Agents,enge by disease or medical therapy, . can develop into a virulent pathogen. For many years this microorganism has gained increasing prominence as an opportunistic pathogen in hospital-associated infections and in compromised patients. Recently, this yeast has been documented as an agent in the morbi

admission

admission

Immune Response to , in Human and Animal Hosts,th the natural host, humans, and in experimental animals have indicated the existence of an active interplay between host defenses and the microorganism’s capacity to evade such defenses. From the standpoint of immunology, we have passed from the mere description of the tissue reactions to the study

FRET

Morphogenetic Transformation of Fungi,ommon to find that the morphology of the fungus in the infected tissue is different from that of the propagule that initiated the infection. It is generally assumed, but not well established experimentally, that the morphogenesis accompanying the parasitic infection in some way confers a survival ad

lymphoma

Melanins and Their Importance in Pathogenic Fungi,rtain yellow, red, green, purple, or blue pigments have similar chemical structures and occasionally are referred to as types of melanins. Melanins are found in humans and various other warm- and cold-blooded vertebrates; invertebrates, including insects; higher plants; fungi; and bacteria, includin

micronized

Cytochrome P450 of Fungi: Primary Target for Azole Antifungal Agents,ane-bound enzyme systems. Such disturbance is caused by the depletion of ergosterol and the accumulation of 14-methylsterols such as lanosterol, 24-methylene-24,25-dihydrolanosterol, obtusifoliol, and 14-methylfecosterol in the membrane. Ergosterol is synthesized from lanosterol with the 14-methylst