Titlebook: Current Review of Cerebrovascular Disease; Marc Fisher (Professor of Neurology),Julien Bogous Book 2001Latest edition Current Medicine, In

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Book 2001Latest editionw of Cerebrovascular Disease provides an update on these rapidly evolving topics and a gives the reader insight into the thought-provoking issues in stroke neurology that have undergone tremendous changes during the past two years. The volume covers four main sections: basic science, diagnostics, cl

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Case Studies and Classroom Tipswe examine the available evidence in support of the following hypothesis: after an ischemic stroke, secondary to the occlusion of an intracranial artery, some of the neurons within the ischemic territory will die via the mechanism of apoptosis.

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https://doi.org/10.1007/978-1-4842-2695-7rly in young patients, the reduced use of x-rays is desirable. During the past few years, the administration of contrast agents and development of high-power gradient systems has contributed to major advances in spatial and temporal resolution and the imaged field of view. The popular 3-D contrast M

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Algorithms for Warping of 2-D PAGE Mapses not necessarily mean a causal relationship [4]: vascular lesions of the brain are frequent in patients with Alzheimer’s pathology [4] and probably contribute to their cognitive decline [5]. VaD is now considered to be the core manifestation of a broad spectrum of clinical syndromes. This chapter

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John Min,William M. Shih,Ga?tan Bellotsupply mainly over the surface of the brain. Ischemia is incomplete and the degree of compensatory blood flow determines whether cells die rapidly and form an infarct, or they survive by simply increasing the oxygen extraction from the blood. Although selective injury to vulnerable neurons may occur

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Mechanisms of Neuronal Cell Death After Ischemic Injury to the Brain,we examine the available evidence in support of the following hypothesis: after an ischemic stroke, secondary to the occlusion of an intracranial artery, some of the neurons within the ischemic territory will die via the mechanism of apoptosis.

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Vascular Dementia,es not necessarily mean a causal relationship [4]: vascular lesions of the brain are frequent in patients with Alzheimer’s pathology [4] and probably contribute to their cognitive decline [5]. VaD is now considered to be the core manifestation of a broad spectrum of clinical syndromes. This chapter

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Neuroprotective Therapy,supply mainly over the surface of the brain. Ischemia is incomplete and the degree of compensatory blood flow determines whether cells die rapidly and form an infarct, or they survive by simply increasing the oxygen extraction from the blood. Although selective injury to vulnerable neurons may occur

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