Titlebook: Current Concepts in Autoimmunity and Chronic Inflammation; Andreas Radbruch,Peter E. Lipsky Book 2006 The Editor(s) (if applicable) and Th

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Weiling Zhou,Bo Li,Zhongjiang Yan,Mao Yangion and uptake of apoptotic and necrotic cells is complex and failures in this process can contribute to the pathogenesis of autoimmune diseases such as systemic lupus erythematosus (SLE). Here, we discuss the recognition and uptake molecules which are involved in an efficient clearance of dying cel

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https://doi.org/10.1007/978-3-030-53614-5e relative ease of staining samples, even at the height of the inflammatory process. These studies led to the study of synovial cytokine regulation, and the identification of TNF as a therapeutic target, which has been amply validated in clinical trials and now routine therapy. The next key question

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Leni Van Goidsenhoven,Anneleen Masscheleinof B cells in the maintenance of the disease process beyond just being precursors of (auto)antibody-producing plasma cells. Detailed analyses have implicated a number of surface molecules and subsequent downstream signalling pathways in the regulation of the events induced by BCR engagement. In this

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S. Rahul,Subin Koshy,R. Sunithape I diabetes (T1D). With proper animal models, some of the complexity behind these diseases can be reduced. The use of linkage analysis and positional cloning of genes in animal models for RA resulted in the identification of one of the genes regulating severity of arthritis in rats and mice, the .

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https://doi.org/10.1007/3-540-29714-6T cell; apoptosis; autoimmune disease; autoimmunity; cell; cytokine; diseases; genes; genetics; infection; mac

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978-3-642-06745-7The Editor(s) (if applicable) and The Author(s), under exclusive license to Springer-Verlag GmbH, DE

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Andreas Radbruch,Peter E. LipskyDiscusses the complex ethiopathologies of autoimmune diseases.Explores the broad variety of cell types and genes involved.Shows how initiating factors in autoimmune disease can possibly be distinct fr

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