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Titlebook: Current Aspects of Blood Coagulation, Fibrinolysis, and Platelets; Ming-Ching Shen (Professor and Chairman),Che-Ming Conference proceedin

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發(fā)表于 2025-3-21 16:56:19 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書目名稱Current Aspects of Blood Coagulation, Fibrinolysis, and Platelets
編輯Ming-Ching Shen (Professor and Chairman),Che-Ming
視頻videohttp://file.papertrans.cn/242/241124/241124.mp4
圖書封面Titlebook: Current Aspects of Blood Coagulation, Fibrinolysis, and Platelets;  Ming-Ching Shen (Professor and Chairman),Che-Ming  Conference proceedin
描述The first Sino-Japanese symposium on Coagulation, Fibrinolysis and Platelets, originating from the Hamamatsu symposium, held in early 1990, was held in Hanchow, Mainland China in October, 1990. During that meeting, Taipei was selected as the next meeting place. We took great pleasure in carrying out this noble endeavor and were honored to have Professor Stenflo, Professor Hedner from Sweden, Professor Aoki and many other distinguished scientists from Japan joining us on this Second Sino-Japanese symposium. Our only regret was that our friends from Mainland China did not attend. The topics covered during the 3-day symposium included areas of both basic and clinical research. The symposium has been an excellent opportunity for all the participants to exchange ideas and improve their expertise, and at the same time, to expand frontiers in the field of coagulation, fibrinolysis and platelets. The symposium was very successful both in terms of contributing greatly to raise the level of research and development in the field of thrombosis and hemostasis and in helping to promote international scientific cooperation and friendship. We would like to express our appreciation to the members o
出版日期Conference proceedings 1993
關(guān)鍵詞coagulation; fibrinolysis; platelets; protase inhibitor; snake venom; amyotrophic lateral sclerosis (ALS)
版次1
doihttps://doi.org/10.1007/978-4-431-68323-0
isbn_softcover978-4-431-70123-1
isbn_ebook978-4-431-68323-0
copyrightSpringer Japan 1993
The information of publication is updating

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發(fā)表于 2025-3-21 22:24:42 | 只看該作者
Titanomagnetite Ore in the Chiney Plutonforms a one to one complex with thrombin to inactivate its procoagulant property. This complex catalyzes activation of protein C and activated protein C inactivates coagulant factors Va and VIIIa in the presence of protein S. Thus, TM converts procoagulant thrombin to an anticoagulant and plays a ce
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地板
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發(fā)表于 2025-3-22 11:15:44 | 只看該作者
Bronislav Gongalsky,Nadezhda Krivolutskayan plasma the activity of such proteases are controlled by specific inhibitors belonging to the serine protease inhibitor (SERPIN) family that forms stoichiometric tight complexes with the target proteases. The inhibitors of the SERPIN family are thought to have a common tertiary structure[1] with a
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發(fā)表于 2025-3-22 15:43:40 | 只看該作者
https://doi.org/10.1007/978-3-030-03880-9tion of factor Xa in the prothrombinase complex (3). Teng and Seegers have also reported that the activation of prothrombin was inhibited by a protein from . venom through its binding to factor Xa (4). We have developed a simple procedure of affinity chromatography on a column of insoluble factor X-
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發(fā)表于 2025-3-22 18:11:29 | 只看該作者
Look! A Squirrel!: Animals Writing,some transglutaminase-inhibitors were found to inhibit the coagulation. We have purified the clottable proteins (CP) from the hemolymph of marine shrimps . and . by DEAE-ion exchanger and gel-filtration. The purified CP was cross-linked to form an insoluble clot . by the shrimp hemocyte transglutami
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發(fā)表于 2025-3-23 00:37:14 | 只看該作者
Wrinkling on Covalently Anchored Hydrogelsis complication. However, the management of bleeding in these patients is a major problem. Low responders can be treated effectively with high doses of clotting factor, whereas treatment is difficult when the clotting factor inhibitor titer is high. Therefore, the diagnosis of the presence of clotti
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發(fā)表于 2025-3-23 02:06:17 | 只看該作者
Wrinkling on Covalently Anchored Hydrogels heterogeneous and patients are categorized into severe, moderate and mild hemophiliacs according to the levels of their functional factor VIII. Approximately 1/3 of the cases are due to . mutations.. Factor VIII is important for normal hemostasis in that it participates in the intrinsic pathway of
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發(fā)表于 2025-3-23 09:06:09 | 只看該作者
Chya-Yan Liaw,Jorge Pereyra,Murat Guvendirennchoconstriction (1). In earlier papers, we reported that PAF may play important roles in the pathogenesis of anaphylactic shock in mice and endotoxin shock and DIC in rats using the first PAF antagonist, CV-3988 (2–5).
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