Titlebook: Copper and Zinc in Inflammatory and Degenerative Diseases; K. D. Rainsford,R. Milanino,G. P. Velo Book 1998 Springer Science+Business Medi

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The biological chemistry of zinc,h the isolation and identification of carbonic anhydrase as a zinc enzyme, its biochemical importance was only recognized in 1939 [2]. The delay in the development of the bioinorganic chemistry of zinc as a field, compared with iron and copper, is attributable to its spectroscopic’ silence’. While c

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Copper complexes for therapy of cancer and autoimmune diseases, treating cancers and autoimmune diseases [see References 1–8 and references cited therein]. Plasma copper concentration increases in neoplastic and autoimmune diseases as an immune-mediated physiological response to these disease states. Treatment with copper complexes is a therapeutic support of t

consent

Zinc and copper in the treatment of rheumatic diseases,. Relevant to the treatment of the rheumatic diseases, zinc deficiency profoundly disturbs the immune function and the host defense mechanism, resulting in lymphoid atrophy, impaired delayed hypersensitivity and T-helper cell dysfunction [2–5]

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Copper and zinc compounds and cell surface interactions,tory and anti-arthritic agents have been well established, based primarily on a wealth of evidence from studies of dietary copper and zinc deficiency and supplementation in laboratory animals and man [1–5]. Surprisingly few studies have, however, focused on the mechanisms of action of copper and zin

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Copper and postmenopausal osteoporosis,lity and consequent risk of fracture [1]. It is the most common metabolic bone disease of postmenopausal women in the Western world. Like other diseases of unknown causation, the aetiology of osteoporosis is considered to be multifactorial. Age, genetic and reproductive history, body weight and vari