Titlebook: Contemporary Oral Oncology; Biology, Epidemiolog Moni Abraham Kuriakose Book 2017 Springer International Publishing Switzerland 2017 Cancer

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全等

親屬

Flu表流動(dòng)

Aetiology of Oral Cavity Cancer,can progress from normal cells to pre-malignant or potentially malignant cells that have the ability to proliferate in a less-controlled fashion than normal [2]. Consequently, the cells become autonomous and cancer results.

滋養(yǎng)

Histopathology of Oral Cavity Cancer and Potentially Malignant Disorders,on between various specialties (surgeon, dentist, medical oncologist, radiation oncologist, pathologist) is as critical for the correct diagnosis, staging, and accurate characterization of the tumor essential for the appropriate management with the best chance for cure.

滋養(yǎng)

Role of Cancer Stem Cells in Oral Cancer,esence of CSCs. This work was done by John Dick and co-workers who successfully demonstrated the critical property of stem cell –self-renewal in association with a cell fraction that constituted only about 1 % of the tumor but were the only cells capable of generating a new tumor in immunocompromised mouse models [2].

異端

The Baroque in Southern and Western Europe,nd Pacific regions (e.g., Papua New Guinea) [2]. It is the most common cancer among males and overall the 3rd most common cancer in India with age-standardized rate (ASR) of 7.2 but contributes highest to the number of new cases annually [3, 4].

LUDE

Prevention of Oral Cancer,nd Pacific regions (e.g., Papua New Guinea) [2]. It is the most common cancer among males and overall the 3rd most common cancer in India with age-standardized rate (ASR) of 7.2 but contributes highest to the number of new cases annually [3, 4].

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