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Titlebook: Connective Tissue Disease; A Comprehensive Guid Dario Roccatello,Lorenzo Emmi Book 2016 Springer International Publishing Switzerland 2016

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發(fā)表于 2025-3-21 20:09:46 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書目名稱Connective Tissue Disease
副標(biāo)題A Comprehensive Guid
編輯Dario Roccatello,Lorenzo Emmi
視頻videohttp://file.papertrans.cn/236/235629/235629.mp4
概述Presents the latest research findings and treatment trends for SLE and Sj?gren syndrome.Covers both basic science and clinical medicine.Discusses “treat to target” interventions.Highlights key points
叢書名稱Rare Diseases of the Immune System
圖書封面Titlebook: Connective Tissue Disease; A Comprehensive Guid Dario Roccatello,Lorenzo Emmi Book 2016 Springer International Publishing Switzerland 2016
描述In this volume devoted to systemic lupus erythematosus (SLE) and Sj?gren syndrome, leading specialists from different disciplines present the latest research findings on many aspects of the diseases and describe the most recent trends in treatment, such as the “treat to target” approach. Both basic science and clinical medicine are addressed, with emphasis on the most promising clinical and laboratory-based studies. The coverage is comprehensive in scope, encompassing, for example, epidemiology, pathogenesis, autoantibodies and biomarkers, disease manifestations, involvement of different organs or systems, relationships with other disorders, biological therapy of SLE, and systemic treatment of Sj?gren syndrome. Readers will find this first volume of Connective Tissue Disease to be an excellent source of information on the current understanding of, and clinical approach to, SLE and Sj?gren syndrome that clearly conveys the progress made in recent years.
出版日期Book 2016
關(guān)鍵詞Autoantibodies; Connective tissue disease; Sj?gren syndrome; Systemic lupus erythematosus; Undifferentia
版次1
doihttps://doi.org/10.1007/978-3-319-24535-5
isbn_softcover978-3-319-79642-0
isbn_ebook978-3-319-24535-5Series ISSN 2282-6505 Series E-ISSN 2283-6403
issn_series 2282-6505
copyrightSpringer International Publishing Switzerland 2016
The information of publication is updating

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https://doi.org/10.1007/BFb0057592s. The disease is characterized by a heterogeneous clinical presentation, a different course in different individuals, and a variability in the disease progression/fluctuations within the same patient. The clinical picture of SLE is extremely variable and may be related to disease activity, organ da
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Nonlinear Transport of the Electron Crystal,estation of the disease, whereas during the course of SLE, it is almost always present. Articular signs include arthralgia and/or arthritis. Arthralgia, which is defined as the presence of joint pain in the absence of clear synovitis, erosion, or deformities, is very frequent both at the onset and d
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https://doi.org/10.1007/BFb0005617 directed against one or more nuclear components, in particular double-stranded DNA. Frequently, SLE involves the kidneys, leading to particular patterns of mainly glomerular inflammation, categorized by the definition of lupus nephritis (LN). LN is a specific expression of SLE. A loss of cellular i
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https://doi.org/10.1007/BFb0005617rogeneous. Symptoms of neuropsychiatric SLE (NPSLE) range from mild diffuse conditions to acute life-threatening events. Although the underlying mechanisms are still largely unraveled, several pathogenic pathways have been identified, such as antibody-mediated neurotoxicity, vasculopathy due aPL, an
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1 State-space models and response formulae,pregnancy is a common event in these women. A better understanding of the disease, the advances of the treatment, and creation of specialized multidisciplinary groups with experience in autoimmune diseases (involving physicians, obstetricians, pediatricians, and midwives) have led to dramatic improv
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