Titlebook: Concepts in Viral Pathogenesis II; Abner Louis Notkins,Michael B. A. Oldstone Textbook 1986 Springer Science+Business Media New York 1986

AMUSE

書目名稱Concepts in Viral Pathogenesis II影響因子(影響力)




書目名稱Concepts in Viral Pathogenesis II影響因子(影響力)學科排名




書目名稱Concepts in Viral Pathogenesis II網絡公開度




書目名稱Concepts in Viral Pathogenesis II網絡公開度學科排名




書目名稱Concepts in Viral Pathogenesis II被引頻次




書目名稱Concepts in Viral Pathogenesis II被引頻次學科排名




書目名稱Concepts in Viral Pathogenesis II年度引用




書目名稱Concepts in Viral Pathogenesis II年度引用學科排名




書目名稱Concepts in Viral Pathogenesis II讀者反饋




書目名稱Concepts in Viral Pathogenesis II讀者反饋學科排名

legacy

GUILE

Topological Aspects of Four-Manifolds,y infect cells through receptors that are involved in normal host functions. These include the binding of λ phage to the maltose receptor [1] and the recognition of the ferrichrome binding site by bacteriophage ? 80 [2].

aerial

Viral Receptors Serving Host Functionsy infect cells through receptors that are involved in normal host functions. These include the binding of λ phage to the maltose receptor [1] and the recognition of the ferrichrome binding site by bacteriophage ? 80 [2].

avenge

Panther

Panther

GULP

Genetic Engineering of Vaccinia Virus Vectors: Development of Live Recombinant Vaccinesruses were soon demonstrated [5–12]. In this article, I shall outline procedures for using vaccinia virus as a vector, summarize the properties of some recombinant viruses, and consider their potential uses.

fatty-streak

Oncogene Activation by Chromosome Translocation the mechanisms by which their function may be critically altered. In the following sections, current findings and hypotheses will be briefly reviewed, emphasizing our own investigations. The field is moving rapidly, and significant additional data will undoubtedly be available by the time this summary is published.

nuclear-tests

Oncogenes, Growth Factors, and Receptorsned recombinational events) of portions of proto-oncogenes by replication-competent retroviruses [2]. These findings have implied the existence of a subset of normal cellular genes with oncogenic potential.