Titlebook: Complement; Hans J. Müller-Eberhard,Peter A. Miescher Book 1985 Springer-Verlag Berlin Heidelberg 1985 Komplement.bacteria.blood.genetics.

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The Human C3b Receptoroteins serving as receptors be identified,. isolated, and characterized. Recent studies of the first complement receptor to be purified, the human C3b receptor, which is also termed CR1, is the subject of this review.

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Molecular Genetics of the Major Histocompatibility Linked Complement Genesathway, are coded for by genes within the major histocompatibility complex (MHC). This conclusion was based first on the demonstration of linkage between C2 deficiency in humans and certain HLA types (HLA-A10, B 18) by Fu et al. [26]. At about the same time, Allan [1] showed linkage of HLA to varian

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The Human C3b Receptorragments of complement proteins that are generated during activation of the system. As is discussed elsewhere in this volume and in a recent review [15], there may be as many as eight different complement receptors on the various cell types that participate in immune and inflammatory reactions. A co

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Initiation of Complement Activationmely the cleavage of C3 into its two primary fragments C3a and C3b. This reaction can be brought about by a number of enzymes but physiologically it is principally caused by the two homologous enzymes which are generated as a consequence of complement activation. These are the two C3 convertases of