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Titlebook: Comparative Pathophysiology of Circulatory Disturbances; Proceedings of the S Colin M. Bloor Conference proceedings 1972 The Editor(s) (if

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51#
發(fā)表于 2025-3-30 09:59:13 | 只看該作者
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發(fā)表于 2025-3-30 16:32:27 | 只看該作者
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發(fā)表于 2025-3-31 00:31:44 | 只看該作者
Comparative Pathophysiology of Pulmonary Hypertension: Development of a Modelompromised by destruction of lung parenchyma and excessive positive alveolar pressure as in chronic obstructive lung disease, by frank obstruction of vessels, large or small, as in pulmonary thrombo-embolic disease, by obstruction to the outflow of blood as in diseases of the pulmonary veins, mitral
55#
發(fā)表于 2025-3-31 03:58:08 | 只看該作者
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發(fā)表于 2025-3-31 06:59:20 | 只看該作者
Comparative Aspects of Coronary Collateral Circulationk the territories of subbranches of the same artery. These vessels have been found in the normal hearts of man (James, 1961, Spalteholz, 1924, Fulton, 1963, 1965, Paulin, 1967) dogs (Blair, 1961, Moore, 1929, Pianetto, 1939) and pigs (Vastesaeger, 1957, Robbins, 1964).Collaterals can be made visible
57#
發(fā)表于 2025-3-31 10:13:51 | 只看該作者
Conference proceedings 1972nimal models exist in nature whose investigative exploitation might make important contributions to an understanding of human disease, yet they are unknown to medical scientists. Conversely, veterinarians are often not aware of the needs of such models and their recognition is often delayed unduly.
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發(fā)表于 2025-3-31 20:12:56 | 只看該作者
Effect of Exercise on Development of Atherosclerosis in Swinerence of sudden death from coronary atherosclerosis and its relationship with age, sex, race, alcohol consumption and physical activity revealed that strenuous activity was infrequently associated with acute coronary occlusion immediately preceding death (4).
60#
發(fā)表于 2025-3-31 22:16:19 | 只看該作者
Comparative Pathophysiology of Pulmonary Hypertension: Development of a Modelring for the moment the role of the lymphatics in draining away interstitial transudate to dispel such edema, it seems reasonable that one of the responses of the pulmonary arteries will be to protect the capillary bed by developing increased resistance to perfusion.
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