Titlebook: Colour Vision Deficiencies XIII; Proceedings of the t C. R. Cavonius Conference proceedings 1997 Springer Science+Business Media Dordrecht

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Levels of expression of the red, green and green-red hybrid pigment genes in the human retinarelative levels of expression of these genes into mRNA and protein in the retina. The red and green pigment genes exist in head-to-tail arrays: one red pigment gene is found 5′ upstream of one or more green pigment genes. In addition to normal red and green pigment genes, 5′green-red3′ hybrid genes,

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Variety of photopigment genes underlying red-green colour visionh. An important aspect of colour vision genotype is the number and ratio of genes that encode the long- and middle-wave-sensitive cone pigments. It has become standard practice to estimate these by quantitative Southern analysis. This method has many inherent problems that limit its usefulness as a

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Phenotypes of anomalous trichromacytrichromacy. First, theoretical calculations show that relatively small individual differences in wavelengths of peak sensitivity of pigments and/or in optical densities can substantially affect Rayleigh matching, which is the procedure typically used to diagnose different types of anomalous trichro

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Unique hues in heterozygotes for protan and deutan deficienciess. In the case of unique blue and green none of the groups of heterozygotes differed significantly from normals. In the case of unique yellow, at a retinal illuminance of 800 td, carriers of protanomaly made settings at significantly shorter wavelengths than any other group. Carriers of deuteranomal

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Results of clinical colour vision tests of ‘Pigmentfarbenanomale’he anomaloscope. These so-called ‘Pigmentfarbenanomale’ (Heinsius 1959) were in a group of 3785 congenital red–green defective patients. Their performance on conventional tests was: pseudoisochromatic plates: mild or moderate anomaly, mostly deutan; Farnsworth’s Panel D-15 Test: pass; lantern test:

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