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Titlebook: Clues in the Diagnosis of Non-tumoral Testicular Pathology; Manuel Nistal,Pilar González-Peramato,álvaro Serra Book 2017 Springer Internat

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書(shū)目名稱(chēng)Clues in the Diagnosis of Non-tumoral Testicular Pathology
編輯Manuel Nistal,Pilar González-Peramato,álvaro Serra
視頻videohttp://file.papertrans.cn/229/228522/228522.mp4
概述Exclusively focuses on non-tumoral testis lesions.Written by highly experienced clinicians.An essential tool for pathologists, urologists, andrologists and pediatricians who face diagnostic problems
圖書(shū)封面Titlebook: Clues in the Diagnosis of Non-tumoral Testicular Pathology;  Manuel Nistal,Pilar González-Peramato,álvaro Serra Book 2017 Springer Internat
描述This book presents the morphology of different non-tumoral lesions of the testis. By showing the differential diagnosis of each lesion, it offers clinicians vital support with diagnosis and treatment. The book is divided into seven main parts: genetic and developmental pathology of the testis; infertility; vascular pathology of the testis; inflammatory pathology; pathology of the rete testis and epididymis; pathology of the vaginal tunic and paratesticular structures; and a final part dealing with miscellanea. Each chapter includes carefully selected figures and a variety of diagrams highlighting the main characteristics of a specific lesion to facilitate its diagnosis..Based on the authors’ experience with hundreds of biopsies, surgical specimens and autopsies, the book presents material that has been gathered over the past 40 years, providing an essential tool for pathologists, urologists, andrologists and pediatricians who face diagnostic problems..
出版日期Book 2017
關(guān)鍵詞Gonadoblastoid dysblasia; Rete testis; Leydig cells; Epididymis; Infertility
版次1
doihttps://doi.org/10.1007/978-3-319-49364-0
isbn_softcover978-3-319-84145-8
isbn_ebook978-3-319-49364-0
copyrightSpringer International Publishing AG 2017
The information of publication is updating

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Uwe‘Cantner,Jean-Luc Gaffard,Lionel Nestanhibin, synaptophysin, and CD56) although there are differences in the intensity of positivity and the distribution of the expression – focal versus diffuse in some cases. The absence of androgen receptors and expression of INSL3 constitute the most relevant histological differential fact.
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https://doi.org/10.1007/978-3-0348-8908-7ded testes are the result of hypogonadism from fetal life, as can be seen in the little proliferation of germ cells and also in postnatal life coinciding with the mini-puberty due to incomplete transformation of gonocytes in Ad spermatogonia. The Ad spermatogonia are the stem cells, and a low number
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