Titlebook: Clinical Physiology of Sleep; Ralph Lydic,Julien F. Biebuyck Book 1988 American Physiological Society 1988 Clinical.Sleep.anesthesia.arrhy

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Yan-Yan Yin,Le-Xi Zhang,Wei Wangep as an index of whether a specific treatment directly affects it. The concept behind this approach is simple and appealing: if an area of the brain is damaged, or if a brain neurotransmitter system is blocked, and REM sleep decreases, then that area of the brain or that neurotransmitter system mus

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https://doi.org/10.1007/978-3-319-51997-5gulatory functions are substantial and are a general phenomenon in mammals and birds. The sleep phase of the daily rest/activity cycle is associated with decreases in metabolic rate and body temperature. In addition, the onset of sleep at any time of day tends to result in declines in metabolic rate

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The Safety and Security Landscape,any years the reigning research paradigm in physiology has been that of homeostasis. Enunciated by Claude Bernard (6) as “constancy of the internal milieu” and articulated by Walter B. Cannon (11) in ., the concept of homeostasis emphasized conservative and energetic aspects of adaptation: constancy

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https://doi.org/10.1007/978-3-642-38977-1rly research by neurophysiologists, neurochemists, physical chemists, and more recently biophysicists. At least two Nobel Prize winners have directed their attention to possible mechanisms of action of anesthetic agents (3, 16, 25). Many intellectual leaders in various fields of science have questio

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https://doi.org/10.1007/978-1-4614-7599-6Clinical; Sleep; anesthesia; arrhythmia; death; energy; evolution; homeostasis; metabolism; muscle; physiology

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https://doi.org/10.1007/978-3-319-51997-5nd external environments. Physiological homeostasis is dependent on the action of feedback mechanisms. The properties of open-or closed-loop operations are now clear, and corresponding mathematical models have been developed for the quantitative analysis of physiological regulations.