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Titlebook: Clinical Manual of Total Cardiovascular Risk; Neil R. Poulter Book 2009 Springer-Verlag London 2009 cardiovascular.heart.management.obesit

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發(fā)表于 2025-3-21 16:57:50 | 只看該作者 |倒序瀏覽 |閱讀模式
書目名稱Clinical Manual of Total Cardiovascular Risk
編輯Neil R. Poulter
視頻videohttp://file.papertrans.cn/229/228078/228078.mp4
概述Data from latest guidelines compiled into one accessible format.Extensive use of illustrations and tables
圖書封面Titlebook: Clinical Manual of Total Cardiovascular Risk;  Neil R. Poulter Book 2009 Springer-Verlag London 2009 cardiovascular.heart.management.obesit
描述Over the last 10–20 years, there has been an increasing appreciation of the need to manage individual risk factors for cardiovascular disease (CVD) in the context of overall cv risk rather than on the basis of the absolute level of any given risk factor. This approach has given rise to the misnomer “global risk” and gen- ated extensive “l(fā)ip-service” around this more broad-minded approach to managing risk factors and the prevention of CVD. This short book was devised with the idea of providing a practical summary of the rationale for management based on estimated total CV risk and the various methods associated with so-doing. Practical issues are addressed including treatment thresholds and targets for the major risk factors on which we routinely intervene, and a brief descr- tion of the major means of these interventions is provided. Whilst a multifactorial approach to CV prevention is logical and reflects the pathophysiological processes which underpin the formation of athe- sclerosis, the evidence base to guide practice using estimated CV risk (“global risk”) as a threshold for intervention is essentially non-existent. Meanwhile, pending supportive evidence from randomized trials
出版日期Book 2009
關(guān)鍵詞cardiovascular; heart; management; obesity; prevention; risk management; stroke; treatment; vascular disease
版次1
doihttps://doi.org/10.1007/978-1-84800-253-1
isbn_softcover978-1-84800-252-4
isbn_ebook978-1-84800-253-1
copyrightSpringer-Verlag London 2009
The information of publication is updating

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Book 2009ogical and reflects the pathophysiological processes which underpin the formation of athe- sclerosis, the evidence base to guide practice using estimated CV risk (“global risk”) as a threshold for intervention is essentially non-existent. Meanwhile, pending supportive evidence from randomized trials
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Cardiovascular disease prevention: prospects for the future?,
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s been an increasing appreciation of the need to manage individual risk factors for cardiovascular disease (CVD) in the context of overall cv risk rather than on the basis of the absolute level of any given risk factor. This approach has given rise to the misnomer “global risk” and gen- ated extensi
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