Titlebook: Clinical Aspects of O2 Transport and Tissue Oxygenation; K. Reinhart,K. Eyrich Conference proceedings 1989 Springer-Verlag Berlin Heidelbe

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Physiology and Pathophysiology of Pulmonary Gas Exchange centrate on recent advances in our understanding of ventilation-perfusion relationships in the lung, and to discuss how abnormalities in the matching of ventilation and blood flow affect gas exchange in several common types of lung disease.

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Microcirculatory Control of Tissue Oxygenationt restriction by the vascular walls. Due to lipid solubility and the diffusivity of oxygen in tissue fluids, diffusional barriers generally do not exist and oxygen exchange between the blood and the extravascular tissue is, therefore, largely a flow-limited phenomenon. As a consequence, tissue oxyge

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Circulatory Compensation as a Response to Hypoxiaone tries to define terms. Not only are there are several categories of hypoxia, but the degree of severity can vary as well as the question of whether it is an acute or chronic situation. There are differences between species in their re sponse to hypoxia which can become important when one tries t

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Oxygen Transport and Oxygen Uptake on the Summit of Mount Everest the human body tolerates extreme oxygen deprivation. Every physician who works in the intensive care environment, or sees patients with severe lung disease, knows that a common problem is severe hypoxemia. Our hope was that by looking at normal man under conditions of extremely severe oxygen lack,

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Supply Dependency of Oxygen Uptakers to vary directly with total O. delivery (total O. delivery is defined as the product of cardiac output and arterial O. content). The more usual situation is that O. extraction in the periphery varies inversely with O. delivery so that O. uptake remains approximately constant. In these certain pat

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Interaction Between Circulating Granulocytes and Xanthine Oxidase-Derived Oxidants in the Postischemion of ischemic tissue (Granger et al. 1981). Since that time much work has been done to substantiate this hypothesis and to further expand on the pathophysiological alterations associated with ischemia reperfusion (I/R). Figure 1 illustrates that during ischemia, cellular ATP is converted to the ca