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Titlebook: Classical Swine Fever and Related Viral Infections; B. Liess Book 1988 Martinus Nijhoff Publishing, Boston 1988 antibody.biology.developme

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發(fā)表于 2025-3-21 16:52:49 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書(shū)目名稱Classical Swine Fever and Related Viral Infections
編輯B. Liess
視頻videohttp://file.papertrans.cn/228/227133/227133.mp4
叢書(shū)名稱Developments in Veterinary Virology
圖書(shū)封面Titlebook: Classical Swine Fever and Related Viral Infections;  B. Liess Book 1988 Martinus Nijhoff Publishing, Boston 1988 antibody.biology.developme
描述The history of research on hog cholera (HC)/classical swine fever (CSF) can be roughly divided into three phases which are characterized by the methods available at the time for demonstrati ng the causati ve agent. Phase covered the period before the viral etiology of HC was discovered by de Schweinitz and Dorset (1904)*. Thereafter (Phase II) the detection of HC virus (HCV) was accomplished by laborious, time-consuming and costly pig inoculation experiments. This explains the extensive search for methods not only for detection but also for accurate infectivity titration as well as for applicable serological techniques to solve urgent problems concerning the pathogenesis, diagnosis, epidemiology and prophylaxis of HC. It was not before the late fifties that HC research entered Phase III when fluorescent antibody techniques offered not only the means for detection and titration of HCV in porcine cell cultures but also for more intensive research on hog cholera and its virus. And yet, there are a number of questions to be answered, e. g. on the genetic and antigenic relation of HCV to bovine viral diarrhea (BVD) virus. There are indications that Phase IV of HC research will bear the
出版日期Book 1988
關(guān)鍵詞antibody; biology; development; epidemiology; immune response; immunity; infection; molecular biology; morph
版次1
doihttps://doi.org/10.1007/978-1-4613-2083-8
isbn_softcover978-1-4612-9235-7
isbn_ebook978-1-4613-2083-8Series ISSN 0924-5359
issn_series 0924-5359
copyrightMartinus Nijhoff Publishing, Boston 1988
The information of publication is updating

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Book 1988porcine cell cultures but also for more intensive research on hog cholera and its virus. And yet, there are a number of questions to be answered, e. g. on the genetic and antigenic relation of HCV to bovine viral diarrhea (BVD) virus. There are indications that Phase IV of HC research will bear the
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0924-5359 ered, e. g. on the genetic and antigenic relation of HCV to bovine viral diarrhea (BVD) virus. There are indications that Phase IV of HC research will bear the 978-1-4612-9235-7978-1-4613-2083-8Series ISSN 0924-5359
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Causality in Quantitative Approaches,, 9, 11) particle sizes varied from approximately 29 nm to 50 nm. In experiments by Kubin (9), e.g., pigs only seroconverted after being inoculated with filtrates from membranes with a pore size of 35–100 nm.
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Stefan Hunziker,Michael Blankenagelction of the developing foetus. These infections may result in prenatal death, the development of a number of foetal abnormalities, stillbirth and, most important, congenital persistent HC virus infection. The chapter concludes with a discussion of the viral pathogenesis of this disease.
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Matthias Lehnert,Bernhard Miller,Arndt Wonka lack of safety. Advances over the last 20 years in our knowledge of hog cholera virus (HCV) and the immunology of the swine species has created a new basis for the development of several live virus vaccines. These are now considered as being effective and totally safe.
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Matthias Lehnert,Bernhard Miller,Arndt Wonkaed; thus UK banned imports of live pigs from the European continent as early as 1882, because a disease called ‘swine fever’ had been discovered. In Denmark the feeding of pigs on uncooked swill in garbage waste areas was forbidden in 1928.
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