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Titlebook: Chronic Obstructive Pulmonary Disease; A Systemic Inflammat Hiroyuki Nakamura,Kazutetsu Aoshiba Book 2017 Springer Science+Business Media S

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發(fā)表于 2025-3-21 18:49:38 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書(shū)目名稱Chronic Obstructive Pulmonary Disease
副標(biāo)題A Systemic Inflammat
編輯Hiroyuki Nakamura,Kazutetsu Aoshiba
視頻videohttp://file.papertrans.cn/227/226401/226401.mp4
概述Considers chronic obstructive pulmonary disease (COPD) not as a simple inflammation of the lung but as a systemic inflammatory disease.Illustrates some comorbidities and associations with other respir
叢書(shū)名稱Respiratory Disease Series: Diagnostic Tools and Disease Managements
圖書(shū)封面Titlebook: Chronic Obstructive Pulmonary Disease; A Systemic Inflammat Hiroyuki Nakamura,Kazutetsu Aoshiba Book 2017 Springer Science+Business Media S
描述This book considers chronic obstructive pulmonary disease (COPD) not as a simple inflammation of the lung but as a systemic inflammatory disease. Beginning with epidemiological studies, etiology, diagnosis and treatment, it elaborates further, illustrating some comorbidities and associations with other respiratory diseases. As such it provides numerous improved and more comprehensive treatment methods, including drug therapies as well as some non-drug therapies.? There are also chapters describing the pathogenesis, genetic abnormalities and newly discovered pathogenetic mechanisms that are expected to be studied further in the future..Edited and written by pioneering researchers, each chapter summarizes the latest trends, describes future prospects and explores the unresolved and critical questions. Chronic Obstructive Pulmonary Disease - A Systemic Inflammatory Disease is a valuable resource to beginning researchers, physicians engaged in clinical practice, supervisors, and basic researchers whose work includes COPD..
出版日期Book 2017
關(guān)鍵詞COPD; Smoking; Systemic Inflammation; Protease; Anti-Protease; Exercise Therapy; Nutritional Therapy; Home
版次1
doihttps://doi.org/10.1007/978-981-10-0839-9
isbn_softcover978-981-10-9266-4
isbn_ebook978-981-10-0839-9Series ISSN 2509-5552 Series E-ISSN 2509-5560
issn_series 2509-5552
copyrightSpringer Science+Business Media Singapore 2017
The information of publication is updating

書(shū)目名稱Chronic Obstructive Pulmonary Disease影響因子(影響力)




書(shū)目名稱Chronic Obstructive Pulmonary Disease影響因子(影響力)學(xué)科排名




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書(shū)目名稱Chronic Obstructive Pulmonary Disease網(wǎng)絡(luò)公開(kāi)度學(xué)科排名




書(shū)目名稱Chronic Obstructive Pulmonary Disease被引頻次




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沙發(fā)
發(fā)表于 2025-3-21 22:58:08 | 只看該作者
https://doi.org/10.1007/978-981-10-0839-9COPD; Smoking; Systemic Inflammation; Protease; Anti-Protease; Exercise Therapy; Nutritional Therapy; Home
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發(fā)表于 2025-3-22 01:51:43 | 只看該作者
978-981-10-9266-4Springer Science+Business Media Singapore 2017
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發(fā)表于 2025-3-22 05:43:15 | 只看該作者
Reflux Aspiration and Lung Diseasenced chronic inflammatory response evoked by the presence of noxious particles or gases in the airways. The progression of airflow limitation has a tremendous impact on disease severity and overall prognosis. However, other factors, such as symptoms, exacerbations, or comorbidities, also have a cons
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發(fā)表于 2025-3-22 12:27:23 | 只看該作者
Reflux Aspiration and Cystic Fibrosis many cases of COPD are caused by heavy cigarette smoking. However, besides cigarette smoking, occupational exposure to airborne particulates and pollutants are also known to be risk factors for developing COPD. Particularly, the indoor use of biomass fuel has been pointed out as one of the major ri
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S. Müller-Lissner,M. Starlinger,H. R. Koelz structural cellular changes contribute to mucus hypersecretion and destruction of the alveolar walls and a decline in forced expiratory volume in one second (FEV.). At the cellular level, macrophages, T lymphocytes, and neutrophils, driven by cytokines including interleukin-8 (IL-8), gather on the
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