Titlebook: Chronic Lymphocytic Leukemia; Book 2005 The Editor(s) (if applicable) and The Author(s), under exclusive license to Springer-Verlag GmbH,

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書目名稱Chronic Lymphocytic Leukemia影響因子(影響力)




書目名稱Chronic Lymphocytic Leukemia影響因子(影響力)學(xué)科排名




書目名稱Chronic Lymphocytic Leukemia網(wǎng)絡(luò)公開度




書目名稱Chronic Lymphocytic Leukemia網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Chronic Lymphocytic Leukemia被引頻次




書目名稱Chronic Lymphocytic Leukemia被引頻次學(xué)科排名




書目名稱Chronic Lymphocytic Leukemia年度引用




書目名稱Chronic Lymphocytic Leukemia年度引用學(xué)科排名




書目名稱Chronic Lymphocytic Leukemia讀者反饋




書目名稱Chronic Lymphocytic Leukemia讀者反饋學(xué)科排名

博愛家

David Kitchin,Adrian Quark,Jayadev Misrahe cell derivation of B-CLL and provided novel hypotheses concerning its pathogenesis. Here we summarize recent work relevant to these issues and conclude that B-CLLmay be derived from a cell that can be referred to as a marginal zone B cell. Moreover, we propose that the lack of chromosomal translo

catagen

,The University of Chicago—1966–1972, epitopes. It is currently unclearwhether this putative antigen-driven process could occur prior to leukaemic transformation and/or that the precursors were transformed into leukaemic cells at distinct maturational stages. Themutational profile of Ig genes has been shown to be associated with diseas

profligate

The Temporality of European Governance times. V-gene usage, VDJ structure, and gene expression differences in the two subgroups allowinsights into differential pathogenic mechanisms and provide further prognostic information (V3-21 usage, ZAP-70 expression). Most importantly, the . mutation status and genomic abnormalities have been sho

commodity

grieve

New Insights into the Phenotype and Cell Derivation of B Cell Chronic Lymphocytic Leukemia,he cell derivation of B-CLL and provided novel hypotheses concerning its pathogenesis. Here we summarize recent work relevant to these issues and conclude that B-CLLmay be derived from a cell that can be referred to as a marginal zone B cell. Moreover, we propose that the lack of chromosomal translo

grieve

蜈蚣

Genotypic Prognostic Markers, times. V-gene usage, VDJ structure, and gene expression differences in the two subgroups allowinsights into differential pathogenic mechanisms and provide further prognostic information (V3-21 usage, ZAP-70 expression). Most importantly, the . mutation status and genomic abnormalities have been sho

grudging

0070-217X g CLL as a very interesting target of many projects which aim at translating the new and exciting developments of basic science into effective new approaches to the patient. .978-3-642-06450-0978-3-540-29933-2Series ISSN 0070-217X Series E-ISSN 2196-9965

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