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Titlebook: Chronic Inflammation; Mechanisms and Regul Masayuki Miyasaka,Kiyoshi Takatsu Book 2016 Springer Japan 2016 Chronic diseases.Proinflammatory

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發(fā)表于 2025-3-21 18:36:46 | 只看該作者 |倒序瀏覽 |閱讀模式
書目名稱Chronic Inflammation
副標(biāo)題Mechanisms and Regul
編輯Masayuki Miyasaka,Kiyoshi Takatsu
視頻videohttp://file.papertrans.cn/227/226384/226384.mp4
概述Provides the most up-to-date summary of cutting-edge research on chronic inflammation.Clarifies the molecular link between chronic inflammation and various diseases including cancer, cardiovascular di
圖書封面Titlebook: Chronic Inflammation; Mechanisms and Regul Masayuki Miyasaka,Kiyoshi Takatsu Book 2016 Springer Japan 2016 Chronic diseases.Proinflammatory
描述.This book provides readers with the most up-to-date information on cutting-edge research concerning chronic inflammation. We now know that when inflammation becomes chronic, it acts as a strong disease-promoting factor in a variety of disorders including arteriosclerosis, obesity, cancer, and Alzheimer disease. Chronic inflammation is hence called as the “silent killer”; it upsets the body’s homeostatic mechanism insidiously. In spite of these developments, we know very little about the mechanism underlying chronic inflammation. Particularly, we do not know precisely what induces chronic inflammation or what promotes its prolongation in a spatiotemporal framework. Neither do we have clear knowledge about how chronic inflammation destroys various tissues or how it predisposes individuals to many different diseases. To make the situation worse, we have no effective treatment against chronic inflammation.?.Since 2010, two major research programs (CREST and PRESTO) aimed at clarifying the mechanisms underlying chronic inflammation were launched in Japan, and investigators of different research areas with a brilliant track record were selected by their research proposals. Subsequently
出版日期Book 2016
關(guān)鍵詞Chronic diseases; Proinflammatory molecules; Tissue remodeling; Fibrosis; Cytokines; Chemokines; Innate ly
版次1
doihttps://doi.org/10.1007/978-4-431-56068-5
isbn_softcover978-4-431-56769-1
isbn_ebook978-4-431-56068-5
copyrightSpringer Japan 2016
The information of publication is updating

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https://doi.org/10.1007/978-94-6209-296-9LIM proteins in the negative regulation of inflammatory responses. These studies should contribute to the development of new therapeutic tools to control the exaggerated inflammation seen in certain human diseases.
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Reflections on Political Theorylly, we found that the Drosophila larval peptide fraction has strong stimulatory activity on the Toll receptor. These findings and future analyses are likely to provide information on the as yet unclear molecular mechanisms of ‘sterile inflammation’ in mammals.
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Reflections on Political Theorye, a lipid mediator, dynamically regulates both the migration and localisation of bone-resorbing macrophage precursor cells. Furthermore, we identified two distinct functional states of differentiated bone-resorbing macrophages, (‘bone-resorptive [R]’ and ‘nonresorptive [N]’), and found that Th17 ce
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Reflections on Political Theorymaging mass spectrometry (IMS) technologies. Owing to recent improvements in IMS regarding its sensitivity, molecular coverage, and spatial resolution, this imaging technology is expected to be a powerful and practical tool to reveal unexpected dynamics of inflammatory mediators in the tissues invol
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Conditions Favouring Political Theorynd identified important molecules that regulate them. In addition, imaging studies using photoactivatable or photoconvertible fluorescent probes have started to reveal how Tfh cells exit the lymphoid organs to become circulating memory cells. In future studies, in order to investigate directly the i
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