Titlebook: Chromosomal Alterations; Origin and Significa Günter Obe,Adayapalam T. Natarajan Book 1994 Springer-Verlag Berlin Heidelberg 1994 Chromosom

Levelheaded

書目名稱Chromosomal Alterations影響因子(影響力)




書目名稱Chromosomal Alterations影響因子(影響力)學(xué)科排名




書目名稱Chromosomal Alterations網(wǎng)絡(luò)公開度




書目名稱Chromosomal Alterations網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Chromosomal Alterations被引頻次




書目名稱Chromosomal Alterations被引頻次學(xué)科排名




書目名稱Chromosomal Alterations年度引用




書目名稱Chromosomal Alterations年度引用學(xué)科排名




書目名稱Chromosomal Alterations讀者反饋




書目名稱Chromosomal Alterations讀者反饋學(xué)科排名

手勢

Factual

悠然

Synergism and Adaptive Response in the Interaction of Low Dose Irradiation with Subsequent Mutagenio a subsequent higher dose. The initial dose may protect by inducing or priming a repair mechanism. This so called “adaptive response” has been demonstrated by numerous authors using different materials and different end points (for reviews, see Kelsey et al. 1991; Liu et al. 1992; Luckey 1991; Ofte

世俗

Implementing a Full Apache Deployment,r by reaction with water which also leads to the release of a proton. For instance the water radical cations convert into OH radicals and protons. The OH radicals diffuse to the DNA and react mainly with the bases and to a smaller extent with the sugar (Scholes et al. 1960). The organic radicals can

玉米棒子

玉米棒子

Red Sanders: Silviculture and Conservationmbination, chromatid segregation and transcription (for a review see Wang 1985; D’Arpa and Liu 1989). These enzymes catalyse the interconversions between topological isomers of DNA. DNA topoisomerase I (Topo I) breaks and rejoins one strand of the DNA double helix, whereas DNA topoisomerase II (Topo

subordinate

Facilities

Chromatin Structure, Hyperthermia and Repair of UV-Induced DNA Photolesions in Mammalian Cells,atin along nucleoskeleton associated repair enzymes. Perhaps nucleoskeleton-centred repair may be confined only to biological important domains comprising active genes. Results of experiments aimed to discover nucleoskeleton-centred repair point to the existence of such a mechanism for transcriptionally active DNA.

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