Titlebook: Cerebral Hemorrhage; Liang-Fu Zhou,Xian-Cheng Chen,Yi-Cheng Lu Conference proceedings 2008 Springer-Verlag Vienna 2008 aneurysm.brain.brai

ODDS

書目名稱Cerebral Hemorrhage影響因子(影響力)




書目名稱Cerebral Hemorrhage影響因子(影響力)學(xué)科排名




書目名稱Cerebral Hemorrhage網(wǎng)絡(luò)公開度




書目名稱Cerebral Hemorrhage網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Cerebral Hemorrhage被引頻次




書目名稱Cerebral Hemorrhage被引頻次學(xué)科排名




書目名稱Cerebral Hemorrhage年度引用




書目名稱Cerebral Hemorrhage年度引用學(xué)科排名




書目名稱Cerebral Hemorrhage讀者反饋




書目名稱Cerebral Hemorrhage讀者反饋學(xué)科排名

脆弱么

dominant

Deferoxamine reduces brain swelling in a rat model of hippocampal intracerebral hemorrhageorrhage (ICH). There were 2 parts to the experiments in this study. In the first part, male Sprague-Dawley rats received a 10-μL infusion of either packed red blood cells (RBC), lysed RBC, hemoglobin, ferrous iron, or saline, into the hippocampus. In the second part, rats received a 10-μL infusion o

Gustatory

The antioxidant effects of melatonin after intracerebral hemorrhage in ratsr and indirect antioxidant, the objective of this study was to evaluate whether melatonin administration would attenuate oxidative stress, brain edema, and neurological deficits in a rat model of ICH. Animals were assigned into groups consisting of sham (needle trauma), vehicle, and melatonin (15 or

使出神

Poly(ADP-ribose) polymerase activation and brain edema formation by hemoglobin after intracerebral hollowing ICH are not well-understood. We have found that hemoglobin plays an important role in edema development after ICH. In this study, we sought to explore the mechanism of brain edema formation caused by hemoglobin..Hemoglobin was infused into the right basal ganglia of male Sprague-Dawley rats

EVICT

Induction of autophagy in rat hippocampus and cultured neurons by iron this study, we examined whether iron can induce autophagy in the hippocampus and in cultured neurons..For in vivo studies, rats received an infusion of either saline or ferrous iron into the right hippocampus and were killed 1, 3, or 7 days later for Western blot analysis of microtubule-associated

EVICT

Effects of superoxide dismutase and catalase derivates on intracerebral hemorrhage-induced brain injn injuries, especially those associated with ischemia/ reperfusion. In this study, we investigated effects of these enzymatic antioxidants on intracerebral hemorrhage (ICH)-induced brain injury..A total of 65 male Sprague-Dawley rats (300-380g) were divided into a sham group, an untreated ICH group,

厚顏無恥

Metallothionein and brain injury after intracerebral hemorrhage genomics study has shown that brain MT-1 and MT-2 mRNA levels are upregulated following intracerebral hemorrhage (ICH) in rats. Our study examines whether brain MT-1 and MT-2 protein levels are increased after ICH. We also investigated the effect of exogenous MT-1 in perihematomal edema formation .

橢圓

Intracerebral hemorrhage injury mechanisms: glutamate neurotoxicity, thrombin, and Srcse utilization (LCGU) studies performed to address this issue demonstrate increased LCGU within hours around an ICH that is blocked by NMDA and AMPA glutamate receptor antagonists. Local injections of NMDA or AMPA increased LCGU whereas glutamate did not, suggesting an ICH effect on glutamate uptake

大溝

Increase in brain thrombin activity after experimental intracerebral hemorrhage perihematomal zone and examined the role of thrombin in ICH-induced brain tissue loss..There were 2 experiments in this study. In the first part, adult male Sprague-Dawley rats received 100 μL of either autologous whole blood or saline. The rats were killed at 1 h or 24 h later for thrombin activit