Titlebook: Cerebellar Degenerations: Clinical Neurobiology; Andreas Plaitakis Book 1992 Springer-Verlag US 1992 anatomy.cortex.neurobiology.neurophys

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Carolin Rotter,Christoph Bresslerdetailed account of the nosology and epidemiology of these disorders is provided in Chapter 8. Some forms of cerebellar degeneration are obscure entities occurring with extreme rarity (even limited to single families), while others show an appreciable prevalence rate in the general population and, a

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https://doi.org/10.1007/978-3-658-36048-1 have been reviewed previously [1–9]. One important phenotype of hexosaminidase deficiency diseases is cerebellar disorder. The focus of this discussion is the nature of the cerebellar disorder and its relation to the other phenotypes of hexosaminidase deficiency disease. An ancillary question is wh

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Cerebellar Degenerations: Clinical Neurobiology978-1-4615-3510-2Series ISSN 0924-8935

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https://doi.org/10.1007/978-3-658-35815-0ve changes in presynaptic and postsynaptic neurons. This property can be explained by the interdependence of neurons for survival and normal function, which derives from the synaptic mode of neuronal connectivity and the trophic interactions of neurons at the level of the synapse.

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Introduction: The Cerebellum and its Disorders in the Dawn of the Molecular Agelar investigation [1], the Greek physician Herophilus (335–200 BC), known as the “father of anatomy,” is generally credited for recognizing the human cerebellum as a distinct brain division. About two millienna later, Sir Thomas Willis (1621–1675) made comparative anatomical observations drawing att

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