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Titlebook: Cellular and Molecular Toxicology of Nanoparticles; Quaiser Saquib,Mohammad Faisal,Abdulrahman A. Alat Book 2018 The Editor(s) (if applica

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樓主: 小天使
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發(fā)表于 2025-3-25 03:32:30 | 只看該作者
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發(fā)表于 2025-3-25 07:52:18 | 只看該作者
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發(fā)表于 2025-3-25 12:28:00 | 只看該作者
Fundamentals of Optical Microscopy,on release and interactions with proteins are the key factors determining the neurotoxicity of AgNPs. AgNPs target endothelial cells forming the blood-brain barrier, neurons and glial cells and leads finally to oxidative stress-related cell death. In this chapter, we review in detail current data on
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發(fā)表于 2025-3-25 18:00:00 | 只看該作者
https://doi.org/10.1007/978-3-642-15964-0the nano-ecotoxicology field had been carried out. However, there is not any standard technique yet to assess toxicity of nanoparticles (NPs) on different biological systems such as reproductive, respiratory, nervous, gastrointestinal systems, and development stages of aquatic organisms. Specific to
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發(fā)表于 2025-3-25 22:23:24 | 只看該作者
https://doi.org/10.1007/978-3-642-15964-0 mollusks, crustaceans, echinoderms and polychaetes have been used as biological models. The main findings related to AgNP toxicity and marine invertebrates are summarized hereafter. Some cellular mechanisms involving nano-internalization (cellular uptake, distribution and elimination), DNA damaging
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發(fā)表于 2025-3-26 04:11:42 | 只看該作者
https://doi.org/10.1007/978-3-642-15964-0ll as behavioural assay clearly hints that signalling pathway like Notch, Wnt, EGFR etc. get affected due to exposure of nanoparticle. Results from various labs prove that nanoparticle can mediate developmental defect by altering signalling pathways. Since many of the signalling pathways are conserv
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發(fā)表于 2025-3-26 07:28:16 | 只看該作者
Cellular and Molecular Toxicology of Nanoparticles
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發(fā)表于 2025-3-26 10:35:05 | 只看該作者
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Advances in Experimental Medicine and Biologyhttp://image.papertrans.cn/c/image/223103.jpg
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發(fā)表于 2025-3-26 16:53:02 | 只看該作者
https://doi.org/10.1007/978-3-319-72041-8Nanotoxicity; Nanoparticles; Genotoxicity; Cell Death; Apoptosis; Transciptomics
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